Dietary vitamin deficiencies disrupt specific metabolic processes and lead to characteristic clinical conditions. Match each essential vitamin on the left to its corresponding deficiency disorder on the right.
- Vitamin A (Retinol)Night blindness (nyctalopia) resulting from impaired photopigment regeneration in rod cells
- Vitamin C (Ascorbic acid)Scurvy, characterized by collagen degradation, bleeding gums, and delayed wound repair
- Vitamin D (Calciferol)Rickets, characterized by impaired intestinal calcium absorption and bone softening
- Vitamin B1 (Thiamine)Beriberi, characterized by impaired carbohydrate metabolism, nerve damage, and cardiac dysfunction
Answer
Vitamin A pairs with night blindness, Vitamin C pairs with scurvy, Vitamin D pairs with rickets, and Vitamin B1 pairs with beriberi.
Each essential vitamin serves a distinct, non-interchangeable biochemical role: Vitamin A forms retinal rhodopsin (preventing night blindness), Vitamin C enables collagen synthesis (preventing scurvy), Vitamin D facilitates calcium absorption (preventing rickets), and Vitamin B1 supports carbohydrate metabolism in nerve and muscle tissues (preventing beriberi).
Step-by-Step Solution
Key Concept
Essential Vitamin Functions and Clinical Deficiency Pathology
Estimated Time:1m 0s