Question

Difficulty: HardReduction of Risk Potential

A client with end-stage renal disease is recovering in the post-anesthesia care unit (PACU) 22 hours after the surgical creation of a left upper extremity arteriovenous (AV) fistula. The client suddenly reports profound weakness. The nurse notes deep, rapid respirations at 3232 breaths/minute. STAT laboratory results reveal a serum potassium of 6.86.8 mEq/L and an arterial blood gas (ABG) showing pH 7.207.20, PaCO 2_2 2929 mm Hg, and HCO 3−_3^- 1010 mEq/L. Which of the following actions should the nurse prioritize to mitigate the highest risk of mortality for this client?

  1. Initiate continuous electrocardiographic monitoring and prepare to administer intravenous calcium gluconate.Answer
  2. B
    Coach the client to slow their breathing rate and utilize a paper bag to prevent excessive carbon dioxide loss.
  3. C
    Prepare equipment for immediate endotracheal intubation to reverse the uncompensated respiratory acidosis.
  4. D
    Place the client in a flat, supine position with the legs elevated to counteract the reported profound weakness.

Answer

The nurse must prioritize stabilizing the myocardial cell membrane to prevent fatal arrhythmias caused by critical hyperkalemia, which is achieved through continuous cardiac monitoring and the administration of intravenous calcium gluconate.
The client's serum potassium level of 6.8 mEq/L constitutes a medical emergency due to the high risk of fatal cardiac arrhythmias. The absolute priority is to stabilize the myocardial cell membrane with intravenous calcium gluconate while initiating continuous cardiac monitoring. The ABG results and Kussmaul respirations reflect a partially compensated metabolic acidosis typical of end-stage renal disease, which is secondary in acute lethality to the severe hyperkalemia.

Step-by-Step Solution

1
Analyze the laboratory results to identify the most life-threatening abnormality.
The serum potassium level of 6.8 mEq/L indicates severe, life-threatening hyperkalemia.
Hyperkalemia at this critical level poses an immediate risk for fatal cardiac arrhythmias, such as ventricular fibrillation or asystole.
2
Analyze the arterial blood gas (ABG) results and respiratory pattern.
The ABG shows a primary metabolic acidosis (low pH, low HCO3-) with partial respiratory compensation (low PaCO2), evidenced by the client's Kussmaul respirations.
The deep, rapid breathing is a necessary compensatory mechanism to expel carbon dioxide and should not be suppressed.
3
Determine the priority nursing intervention based on the identified risks.
The priority action is to address the severe hyperkalemia by initiating electrocardiographic monitoring and administering calcium gluconate.
While metabolic acidosis is present, the immediate threat to life is cardiac arrest from hyperkalemia. Calcium gluconate acts rapidly to stabilize the resting membrane potential of the myocardium.

Key Concept

Prioritization of care in severe hyperkalemia and interpretation of compensatory Kussmaul respirations in metabolic acidosis.
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