A telemetry nurse is called to the room of a client who suffered a comminuted pelvic fracture seventy-two hours prior. The client is suddenly grasping their chest, expressing a severe feeling of doom, and sweating profusely. Assessment reveals a heart rate of beats per minute, a respiratory rate of breaths per minute, and a blood pressure of . Room air pulse oximetry displays an oxygen saturation of . The rapid arterial blood gas (ABG) results are as follows: , , , and . Which of the following conclusions accurately reflects this client's current physiological derangement?
- Uncompensated respiratory alkalosis with severe hypoxemia, indicating a probable pulmonary embolism.Answer
- BUncompensated respiratory acidosis with hypoxemia, indicating the rapid onset of acute respiratory failure.
- CFully compensated metabolic alkalosis, reflecting the body's compensatory response to acute pain and anxiety.
- DMetabolic acidosis with a widened pulse pressure, indicating early hemorrhagic shock from the pelvic fracture.
Answer
The client is experiencing uncompensated respiratory alkalosis and severe hypoxemia, which strongly suggests a pulmonary embolism.
The arterial blood gas results demonstrate an elevated pH (alkalosis) driven by a decreased PaCO2 (respiratory origin). The normal bicarbonate level indicates there is no metabolic compensation yet. This state of uncompensated respiratory alkalosis, paired with profound hypoxemia (low PaO2), sudden feeling of doom, and severe tachycardia in a client with a recent pelvic fracture, is the classic clinical presentation of a pulmonary embolism causing dead-space ventilation and resultant hyperventilation.
Step-by-Step Solution
Key Concept
Arterial Blood Gas Interpretation and Pulmonary Embolism Recognition