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Zorluk: ZorReduction of Risk Potential

A nurse is caring for an adult client with end-stage cirrhosis who recently underwent an ultrasound-guided bedside paracentesis with the removal of 4.5 liters4.5 \text{ liters} of ascitic fluid. Prior to the procedure, the client's vital signs were blood pressure 126/82 mmHg126/82 \text{ mmHg} and heart rate 86 bpm86 \text{ bpm}.

Thirty minutes post-procedure, the client reports feeling slightly dizzy. The current assessment reveals the following data:

ParameterCurrent Value
Blood Pressure92/74 mmHg92/74 \text{ mmHg}
Heart Rate118 bpm118 \text{ bpm}
Respiratory Rate20 breaths/min20 \text{ breaths/min}
SpO2SpO_296%96\% on room air
Arterial pHpH7.327.32
PaCO2PaCO_231 mmHg31 \text{ mmHg}
HCO3−HCO_3^-15 mEq/L15 \text{ mEq/L}

Which action should the nurse take next to reduce the risk of severe physiological compromise?

  1. A
    Continue routine monitoring, as the widened pulse pressure indicates an expected hemodynamic response to fluid removal.
  2. B
    Apply supplemental oxygen to treat the underlying respiratory acidosis shown on the morning laboratory results.
  3. Initiate the prescribed intravenous infusion of 25% human albumin.Cevap
  4. D
    Elevate the head of the bed to a high Fowler's position to maximize diaphragmatic excursion.

Cevap

The correct action is to initiate the prescribed intravenous infusion of 25% human albumin to prevent paracentesis-induced circulatory dysfunction.
The removal of a large volume of ascitic fluid rapidly decreases intra-abdominal pressure, leading to splanchnic vasodilation and a massive shift of intravascular fluid into the peritoneal space. This phenomenon, known as paracentesis-induced circulatory dysfunction (PICD), causes severe hypovolemia. The client's narrowing pulse pressure (92−74=18 mmHg92 - 74 = 18 \text{ mmHg}) and tachycardia (118 bpm118 \text{ bpm}) are classic early compensatory signs of hypovolemic shock. Administering a hypertonic colloid, such as 25% human albumin, increases intravascular oncotic pressure, pulling fluid back into the vascular space and effectively mitigating the risk of cardiovascular collapse.

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1
Analyze the vital sign changes from baseline.
The blood pressure has dropped significantly to 92/74 mmHg92/74 \text{ mmHg}, the pulse pressure has narrowed to 18 mmHg18 \text{ mmHg} (92−74=1892 - 74 = 18), and the heart rate has increased to 118 bpm118 \text{ bpm}.
Recognizing early hemodynamic trends is essential for detecting the onset of compensatory hypovolemic shock.
2
Interpret the arterial blood gas (ABG) values.
The ABG indicates a metabolic acidosis (pHpH 7.327.32, HCO3−HCO_3^- 1515) with partial respiratory compensation (PaCO2PaCO_2 3131), not a respiratory acidosis.
Accurate ABG interpretation prevents implementing inappropriate interventions, such as attempting to correct a nonexistent respiratory problem.
3
Evaluate the physiological impact of large-volume fluid removal.
The rapid removal of 4.5 liters4.5 \text{ liters} of fluid drastically decreases intra-abdominal pressure, causing splanchnic vasodilation and a massive intravascular fluid shift.
Understanding the fluid dynamics of a large-volume paracentesis highlights the immediate risk for profound hypovolemia.
4
Select the priority risk-reduction intervention.
Administering a hypertonic colloid, such as 25% human albumin, is selected to restore intravascular volume.
Albumin provides the necessary oncotic pressure to pull fluid back into the vascular space, stabilizing hemodynamics and preventing cardiovascular collapse.

Anahtar Kavram

Paracentesis-Induced Circulatory Dysfunction (PICD)
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