A client is admitted to the intensive care unit with diabetic ketoacidosis (DKA). At admission, the client's serum glucose was () and serum potassium was (). The client was started on a continuous intravenous (IV) regular insulin infusion and fluid resuscitation with sodium chloride.
Four hours later, the client's serum glucose is () and serum potassium is (). The anion gap remains elevated at (normal: ). Which of the following modifications to the client's parenteral therapy should the nurse anticipate next?
- Change the continuous IV fluid to include dextrose and add intravenous potassium chloride.Cevap
- BDiscontinue the continuous insulin infusion and administer a subcutaneous dose of rapid-acting insulin.
- CAdminister intravenous sodium bicarbonate to rapidly correct the elevated anion gap acidosis.
- DMaintain the sodium chloride but decrease the IV insulin rate to to prevent hypoglycemia.
Cevap
The nurse should anticipate changing the continuous IV fluid to include dextrose and adding intravenous potassium chloride.
The correct action is to add dextrose to the IV fluids and initiate potassium supplementation. In the treatment of DKA, insulin resolves both hyperglycemia and ketoacidosis. However, blood glucose normalizes much faster than ketones are cleared. When glucose falls below , dextrose must be added to the IV fluids. This provides a carbohydrate source, preventing hypoglycemia while allowing the nurse to keep the insulin infusion running to close the elevated anion gap. Furthermore, insulin shifts potassium into the cells. With a potassium level of and an active insulin drip, potassium replacement is critical to prevent cardiac dysrhythmias.
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Anahtar Kavram
In DKA management, when blood glucose falls below (), IV fluids must be transitioned to include dextrose. This allows the continuation of the IV insulin drip, which is essential to close the anion gap and clear ketones, while preventing dangerous hypoglycemia. Concurrently, potassium must be proactively replaced to counteract insulin-driven intracellular shifts.