Soru

Zorluk: Çok zorEstablishing Priorities

An emergency department nurse is managing a client admitted with severe diabetic ketoacidosis (DKA). The client is lethargic, with a blood pressure of 82/48 mm Hg, heart rate of 128 beats/minute, and Kussmaul respirations. Initial laboratory results reveal a blood glucose of 780 mg/dL (43.3 mmol/L), serum pH of 7.15, and serum potassium of 3.1 mEq/L (3.1 mmol/L). Arrange the following prescribed interventions in the correct chronological sequence of execution from first to last.

  1. 1Insert a large-bore intravenous catheter and administer a rapid bolus of 0.9% sodium chloride.
  2. 2Infuse an intravenous piggyback of potassium chloride.
  3. 3Initiate a continuous intravenous infusion of regular insulin.
  4. 4Add 5% dextrose to the continuous intravenous hydration fluids.
  5. 5Administer a prescribed dose of subcutaneous basal insulin.

Cevap

The correct sequence is: Fluid resuscitation (0.9% NaCl), followed by Potassium replacement, then Intravenous Regular Insulin, later adding 5% Dextrose when glucose drops, and finally Subcutaneous Basal Insulin before discontinuing the drip.
The priority in DKA is always ABCs and perfusion; thus, isotonic fluid resuscitation is the first action for a client with severe hypotension. The critical secondary priority is electrolyte management. Because insulin drives potassium into cells, starting insulin with a baseline potassium of 3.1 mEq/L is highly dangerous. Potassium replacement must precede or accompany the insulin infusion. Once the client is hemodynamically stable and electrolytes are safe, insulin is initiated. As glucose approaches 250 mg/dL, dextrose is added to prevent hypoglycemia while allowing continuous insulin to resolve the acidosis. Finally, subcutaneous insulin is given prior to stopping the drip to maintain baseline glycemic control.

Adım Adım Çözüm

1
Administer 0.9% sodium chloride bolus.
Restores intravascular volume and organ perfusion.
Osmotic diuresis causes profound hypovolemia, which takes precedence over correcting the metabolic derangements.
2
Administer potassium chloride intravenous rider.
Increases serum potassium levels.
Insulin administration drives potassium into the intracellular space. With a baseline potassium of 3.1 mEq/L, administering insulin first would cause profound, lethal hypokalemia.
3
Initiate continuous intravenous regular insulin.
Stops lipolysis and ketogenesis, lowering blood glucose.
Once perfusion and electrolytes are secured, insulin addresses the root pathophysiology of DKA.
4
Add 5% dextrose to intravenous fluids.
Maintains blood glucose between 150-200 mg/dL.
Added when glucose reaches 250 mg/dL to prevent hypoglycemia and cerebral edema, as insulin must continue running until the acidosis (anion gap) resolves.
5
Administer subcutaneous basal insulin.
Provides long-acting glycemic control.
Required to prevent rebound ketoacidosis when transitioning off the intravenous insulin infusion.

Anahtar Kavram

Prioritization in Diabetic Ketoacidosis (DKA) Resuscitation
Bu soruyu puanla