Orthopedic Surgery

132 questions

Question 61Question →

A 34-year-old right-handed male presents to the emergency department one hour after an altercation in which he was struck on the right upper arm with a heavy steel pipe. He complains of intense pain and an inability to use his right hand. His vital signs are within normal limits. On physical examination, the right arm reveals significant soft tissue swelling, ecchymosis, and a visible deformity over the mid-humerus. The skin is completely intact. Neurological examination of the right upper extremity reveals an inability to extend the wrist or fingers. Sensation to pinprick is diminished over the dorsal aspect of the first web space. The radial and ulnar pulses are 2+2+ and symmetric. Capillary refill is less than 22 seconds in all digits. Anteroposterior and lateral radiographs of the right humerus demonstrate a displaced transverse fracture of the middle third of the humeral shaft. Which of the following is the most appropriate next step in management?

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Answer: Application of a coaptation splint and clinical observation

Answer

Application of a coaptation splint and clinical observation
The clinical presentation is consistent with a closed midshaft humeral fracture complicated by a primary radial nerve palsy. The radial nerve courses through the spiral groove on the posterior aspect of the humerus, making it uniquely vulnerable to midshaft fractures. This injury results in weakness of wrist and finger extension (wrist drop) and sensory loss over the dorsal first web space. In the setting of a closed fracture, this is almost always a neurapraxia (stretch injury) rather than nerve transection. Because >90%>90\% of these palsies recover spontaneously within 3 to 4 months, the standard of care is conservative management. This involves immobilizing the fracture with a coaptation splint (or functional brace) and scheduling close outpatient follow-up to monitor for clinical signs of nerve recovery. Surgical exploration is generally reserved for open fractures, associated vascular injuries requiring repair, or a secondary nerve palsy (a deficit that appears only after a closed reduction attempt, suggesting the nerve has become entrapped in the fracture site).

Step-by-Step Solution

1
Identify the primary injury from the physical exam and radiographic findings.
The patient has a closed, displaced transverse fracture of the midshaft humerus.
Establishing the correct anatomical fracture pattern is necessary to anticipate common complications.
2
Analyze the neurological deficits to identify the specific nerve involved.
Inability to extend the wrist/fingers and numbness in the first dorsal web space indicate a radial nerve palsy.
The radial nerve runs along the spiral groove of the mid-humerus, making it highly susceptible to injury in midshaft fractures.
3
Determine the appropriate management for a primary radial nerve palsy in a closed humeral shaft fracture.
Conservative management with a splint is chosen.
Primary radial nerve palsies in closed fractures are almost always neurapraxias that resolve spontaneously in >90%>90\% of cases. Immediate surgery is contraindicated unless specific criteria are met.

Key Concept

Primary radial nerve palsy in the setting of a closed humeral shaft fracture does not require immediate surgical exploration. Initial management is conservative with immobilization (e.g., coaptation splint) and clinical observation.
Question 62Question →

A 31-year-old male agricultural worker presents to the emergency department with a 3-day history of progressive pain and swelling in his left index finger. He reports sustaining a small puncture wound from a wire fence 5 days prior. His temperature is 38.1∘C38.1^\circ\text{C} (100.6∘F100.6^\circ\text{F}), blood pressure is 118/76 mmHg118/76\text{ mmHg}, and pulse is 98/min98/\text{min}. Physical examination reveals a diffusely swollen, erythematous left index finger that the patient holds in a partially flexed posture. There is pronounced tenderness extending along the volar aspect of the digit down to the proximal palmar crease. Passive extension of the finger elicits excruciating pain. Sensation and capillary refill are intact in the affected digit. Radiographs of the hand show soft tissue swelling but no foreign body, fracture, or bony destruction. Which of the following is the most appropriate next step in management?

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Answer: Immediate surgical irrigation and debridement of the flexor tendon sheath

Answer

Immediate surgical irrigation and debridement of the flexor tendon sheath is the correct next step because the patient has acute suppurative flexor tenosynovitis, which is a surgical emergency.
The correct answer addresses the urgent need for surgical decompression in acute suppurative flexor tenosynovitis. The patient presents with the four classic Kanavel signs: 1) fusiform (sausage-like) swelling of the digit, 2) the digit is held in a flexed resting posture, 3) exquisite tenderness along the course of the flexor tendon sheath, and 4) severe pain elicited by passive extension of the finger. Because the flexor tendon sheath is a closed space with a tenuous blood supply, the accumulation of purulent material rapidly increases pressure, causing ischemia and necrosis of the tendon. Therefore, immediate surgical irrigation and debridement in the operating room, combined with intravenous antibiotics, is the definitive and mandatory next step to preserve digit function.

Step-by-Step Solution

1
Analyze the patient's history and physical examination findings.
The patient has a history of a puncture wound followed by fusiform swelling, a flexed resting posture, tenderness along the tendon sheath, and severe pain with passive extension.
Identifying these specific clinical features is necessary to recognize the four classic Kanavel signs.
2
Synthesize the physical findings to establish a diagnosis.
The presence of Kanavel signs confirms the clinical diagnosis of acute suppurative flexor tenosynovitis.
This specific pattern of infection behaves like compartment syndrome of the digit due to the closed space of the synovial sheath.
3
Determine the urgency and appropriate intervention based on the diagnosis.
Recognize this as a surgical emergency requiring immediate operative irrigation and debridement alongside intravenous antibiotics.
Delaying surgery or relying solely on antibiotics leads to sheath ischemia, tendon necrosis, and permanent loss of digit function.

Key Concept

Acute suppurative flexor tenosynovitis diagnosis and management
Estimated Time:1m 15s
Question 63Question →

A 29-year-old woman is brought to the trauma center after being thrown from her motorcycle in a high-speed collision. The primary trauma survey reveals a patent airway, unlabored breathing, and intact circulation. Her vital signs are within normal limits. The secondary survey is notable for a severe right lower extremity injury. There is an obvious deformity of the midshaft tibia with a 14-cm full-thickness laceration. The underlying muscle is severely crushed, and exposed bone is stripped of its periosteum. The dorsalis pedis and posterior tibial pulses are palpable. Arrange the following steps in the most appropriate chronological sequence for the optimal management of this patient's limb injury.

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Answer

The optimal sequence is: 1) Intravenous antibiotics and tetanus prophylaxis, 2) Bedside realignment and splinting, 3) Surgical debridement in the OR, 4) Skeletal stabilization with an external fixator, and 5) Delayed soft tissue reconstruction.
The management of severe open fractures follows a strict protocol prioritizing infection prevention and sequential tissue management. Immediate systemic antibiotics represent the most critical first step, followed by temporary bedside stabilization. In the operating room, thorough debridement must precede the placement of any stabilization hardware. Finally, highly contaminated wounds with extensive soft tissue loss (Gustilo IIIB) require serial evaluations and delayed definitive soft tissue coverage.

Step-by-Step Solution

1
Administer IV antibiotics and tetanus prophylaxis
Systemic antimicrobial coverage is established
Early antibiotic administration (within 1 hour) is the single most important intervention in decreasing infection rates for open fractures.
2
Perform bedside realignment and temporary splinting
The limb is stabilized and the wound is covered
Reduces tension on compromised soft tissues, relieves pain, and protects neurovascular structures pending definitive surgical intervention.
3
Execute radical surgical debridement
Contaminants and devitalized tissues are removed
A clean, viable wound bed is a prerequisite for any hardware placement and prevents deep-seated bone infections.
4
Apply an external fixator
Rigid skeletal stability is achieved
Stabilizes the fracture without introducing large hardware into a highly contaminated field, facilitating subsequent wound care and healing.
5
Perform soft tissue flap reconstruction
Exposed bone and neurovascular structures are permanently covered
Requires a completely sterile wound bed, necessitating a deliberate delay (usually 3-7 days) and serial debridements before execution.

Key Concept

Sequential management of severe high-energy open fractures (Gustilo-Anderson Type IIIB)
Question 64Question →

A 68-year-old man presents to the emergency department with a 2-day history of worsening right knee pain, swelling, and fever. He underwent a primary right total knee arthroplasty 3 weeks ago for severe osteoarthritis. His early postoperative course was uncomplicated, and he had been progressing well with physical therapy until the onset of these symptoms. His temperature is 38.9°C (102.0°F), blood pressure is 125/80 mmHg, pulse is 105/min, and respiratory rate is 18/min. Physical examination reveals a markedly swollen, erythematous, and warm right knee. The surgical incision is well-healed with no active drainage. Range of motion is severely limited by pain. Laboratory studies show a leukocyte count of 14,500/mm³ and an erythrocyte sedimentation rate of 85 mm/hr. Arthrocentesis of the right knee yields cloudy fluid with a leukocyte count of 45,000/mm³ (90% neutrophils). Gram stain shows Gram-positive cocci in clusters. Which of the following is the most appropriate next step in the management of this patient?

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Answer: Surgical debridement, polyethylene exchange, and retention of the metal components

Answer

Surgical debridement, polyethylene exchange, and retention of the metal components
The correct answer is surgical debridement, polyethylene exchange, and retention of the metal components (Debridement, Antibiotics, and Implant Retention or DAIR). This patient presents with an acute early-onset prosthetic joint infection (PJI), defined as occurring within 4 weeks of the primary arthroplasty. The standard of care for acute early PJIs or acute hematogenous infections (with < 3 weeks of symptoms) in a well-fixed prosthesis is the DAIR procedure. This involves open arthrotomy, thorough washout, exchange of modular components (like the polyethylene insert to remove any formed biofilm), and retention of the fixed metal implants, followed by a prolonged course of targeted intravenous antibiotics.

Step-by-Step Solution

1
Identify the primary diagnosis based on the clinical presentation and arthrocentesis results.
The patient has an acute early-onset prosthetic joint infection (PJI) caused by Staphylococcus species (Gram-positive cocci in clusters), indicated by fever, joint pain, and synovial fluid pleocytosis 3 weeks post-surgery.
Accurate diagnosis is required to dictate the surgical algorithm.
2
Evaluate the timeframe of the infection relative to the primary surgery.
The infection onset is at 3 weeks post-operation, placing it in the 'acute early' category (defined as less than 4 weeks post-operatively).
The chronicity of the infection determines whether the prosthetic hardware must be completely removed or can be safely retained.
3
Select the definitive management strategy based on the acute timeframe.
Debridement, antibiotics, and implant retention (DAIR) is chosen over a two-stage complete revision.
In acute early PJIs, the bacterial biofilm is immature. Thorough washout with exchange of modular parts (polyethylene insert) combined with IV antibiotics is sufficient to eradicate the infection while preserving the fixed metal hardware.

Key Concept

The management of acute early-onset prosthetic joint infections (< 4 weeks post-op) involves Debridement, Antibiotics, and Implant Retention (DAIR), rather than complete removal of the prosthesis.
Question 65Question →

Match each clinical presentation of an acute orthopedic injury to its most likely associated neurological examination deficit.

Click a left item, then click its matching right item

Items

A 42-year-old male falls forcefully on his shoulder; examination reveals a flattened anterior shoulder contour and an externally rotated, slightly abducted arm.
A 28-year-old male involved in a motor vehicle collision presents with severe hip pain; his right lower extremity is shortened, internally rotated, and adducted.
A 35-year-old female falls on an outstretched hand, sustaining a supracondylar humerus fracture; radiographs show anteromedial displacement of the proximal fracture fragment.
A 22-year-old male sustains a direct blow to the lateral aspect of the leg, resulting in a displaced fracture of the fibular neck.
A 31-year-old male sustains a transverse midshaft humerus fracture after being struck with a heavy object.

Matches

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Answer

Anterior shoulder dislocations correspond to axillary nerve deficits; posterior hip dislocations to sciatic nerve deficits; anteromedially displaced supracondylar fractures to median nerve deficits; fibular neck fractures to common peroneal nerve deficits; and midshaft humerus fractures to radial nerve deficits.
Each correct pair accurately links a classic orthopedic injury mechanism to the specific peripheral nerve that is anatomically most vulnerable at that site, and subsequently matches that nerve to its exact motor and sensory distribution.

Step-by-Step Solution

1
Identify the specific fracture or dislocation pattern presented in each clinical vignette.
The vignettes describe anterior shoulder dislocation, posterior hip dislocation, supracondylar humerus fracture, fibular neck fracture, and midshaft humerus fracture.
Recognizing the exact anatomical location and mechanism of the injury is required to determine the structures at risk.
2
Determine the neurovascular structure that runs in closest proximity to the injured bone or joint.
The associated nerves are the axillary nerve, sciatic nerve, median nerve, common peroneal nerve, and radial nerve, respectively.
Acute orthopedic injuries predictably threaten adjacent peripheral nerves due to traction, compression, or direct laceration.
3
Correlate the injured peripheral nerve with its expected motor and sensory examination findings.
Matched the anatomical nerve injury to specific physical exam deficits (e.g., matching the common peroneal nerve to foot drop and dorsal sensory loss).
Each peripheral nerve has a distinct innervation pattern that dictates the resulting clinical neurological deficit.

Key Concept

Predictable peripheral nerve injuries associated with specific acute orthopedic fractures and joint dislocations.
Question 66Question →

During morning rounds, the orthopedic team evaluates a 24-year-old male who was admitted overnight following a closed mid-shaft right tibia fracture sustained during a soccer match. He was treated with closed reduction and application of a long-leg cast 88 hours ago. The patient is now highly agitated, reporting severe, throbbing pain in his right leg that has worsened significantly despite escalating doses of intravenous hydromorphone. His medical history is notable for opioid use disorder, for which he takes daily methadone. Vital signs are within normal limits. Physical examination reveals a pink, warm right foot with a capillary refill time of 22 seconds. Dorsalis pedis and posterior tibial pulses are easily palpable. However, passive plantar flexion of the toes elicits excruciating pain in the anterior shin, and the patient reports decreased sensation in the first dorsal web space. Which of the following is the most appropriate next step in management?

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Answer: Bivalve the cast and divide the underlying padding down to the skin

Answer

Bivalve the cast and divide the underlying padding down to the skin
The correct response identifies that the immediate first step for suspected acute compartment syndrome (ACS) in a patient with a cast is to remove all external constriction. Bivalving the cast and dividing the underlying padding down to the skin can drop compartment pressures dramatically (by up to 85%85\%). This action is mandatory before proceeding to invasive pressure measurements or definitive surgical fasciotomy.

Step-by-Step Solution

1
Analyze the patient's symptoms and physical exam findings.
The patient has pain out of proportion to the injury, severe pain with passive stretch (plantar flexion), and early nerve ischemia (decreased sensation in the first web space).
These findings are the classic, cardinal signs of acute compartment syndrome (ACS), which requires immediate recognition.
2
Interpret the vascular status (palpable pulses and brisk capillary refill).
Vascular status is currently intact.
Intracompartmental pressure required to cause ischemia is lower than arterial pressure, meaning pulses will remain palpable until extremely late in the course of ACS. Pulses do not rule out the diagnosis.
3
Determine the algorithmic next step for suspected ACS in a casted extremity.
The absolute first step is the release of all external circumferential constriction.
Bivalving a cast and cutting the underlying webril/padding down to the skin can reduce intracompartmental pressures by 50%50\% to 85%85\%, potentially preventing the need for surgical fasciotomy.

Key Concept

The immediate management of suspected acute compartment syndrome in a casted or splinted extremity.
Estimated Time:2m 0s
Question 67Question →

A 5-year-old girl is brought to the emergency department with a 24-hour history of high fever (39.2∘C39.2^\circ\text{C} or 102.6∘F102.6^\circ\text{F}) and refusal to bear weight on her right leg. On examination, her right hip is exquisitely tender and held rigidly in flexion, abduction, and external rotation. Any attempted passive range of motion elicits severe distress. Assuming all subsequent diagnostic findings support a diagnosis of acute septic arthritis, arrange the following clinical management steps in the most appropriate chronological sequence.

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Answer

The correct chronological sequence is: 1) Obtain plain radiographs and labs, 2) Perform a diagnostic ultrasound, 3) Perform an image-guided arthrocentesis, 4) Initiate empiric intravenous antibiotics, 5) Proceed with emergent surgical arthrotomy.
The standard sequence for evaluating and managing a child with a suspected septic hip prioritizes non-invasive broad diagnostics followed by targeted, invasive interventions. Initial plain radiographs and blood work rule out overt bony pathology and establish the pre-test probability via the Kocher criteria. Following this, an ultrasound is the safest and most sensitive tool to confirm a joint effusion. Once an effusion is found, diagnostic arthrocentesis is the critical next step; crucially, fluid for culture must be aspirated before the administration of antibiotics to ensure accurate pathogen identification. After cultures are drawn, empiric intravenous antibiotics are initiated immediately. Finally, because the hip joint is a confined space where purulence can cause vascular tamponade and subsequent avascular necrosis of the femoral head, emergent surgical arthrotomy and lavage is the definitive treatment.

Step-by-Step Solution

1
Analyze the clinical presentation.
The patient has symptoms highly suspicious for pediatric septic arthritis of the hip (fever, non-weight-bearing, hip held in flexion/abduction/external rotation).
Identifying the suspected diagnosis establishes the need for an urgent, step-wise diagnostic and therapeutic algorithm.
2
Determine the first step in the diagnostic workup.
Obtain plain radiographs and baseline serum inflammatory markers.
Plain radiographs are essential to initially rule out other structural causes of pediatric hip pain (e.g., fracture, slipped capital femoral epiphysis), while markers (ESR, CRP, WBC) establish the pre-test probability based on Kocher criteria.
3
Select the appropriate advanced imaging modality.
Perform a diagnostic ultrasound of the hip.
Ultrasound is highly sensitive and non-invasive for detecting a joint effusion, which confirms the presence of intra-articular fluid before attempting an invasive needle aspiration.
4
Determine the optimal timing of fluid sampling.
Perform image-guided arthrocentesis.
Aspiration of the joint fluid for cell count, Gram stain, and culture must occur before any antimicrobial therapy is administered to prevent sterilizing the sample.
5
Establish the timing of pharmacological therapy.
Initiate empiric intravenous antibiotics.
Once cultures are successfully secured, broad-spectrum antibiotics should be started immediately to prevent further systemic spread and joint destruction while the operating room is prepared.
6
Identify the definitive surgical management.
Emergent surgical arthrotomy and joint lavage.
The hip is a tight space; purulence under pressure can quickly cause tamponade of the retinacular vessels, leading to avascular necrosis. Surgical decompression is therefore mandatory.

Key Concept

The chronological management of suspected pediatric septic arthritis emphasizes structural rule-out, confirming the presence of an effusion, securing cultures via aspiration prior to antibiotic administration, and culminating in emergent surgical decompression.
Estimated Time:1m 30s
Question 68Question →

An otherwise healthy 7-year-old boy arrives at the trauma bay 45 minutes after falling from a playground structure onto his outstretched right hand. He complains of excruciating elbow pain and refuses to move his arm. Vital signs are within normal limits for his age. On physical examination, the right elbow is markedly swollen with an S-shaped deformity. The skin over the antecubital fossa is intact, taut, and slightly ecchymotic. Neurologic evaluation reveals an inability to flex the distal interphalangeal joints of the index finger and thumb to make an "OK" sign, though wrist extension and finger abduction are intact. The radial pulse is non-palpable and cannot be detected with a handheld Doppler ultrasound. However, the right hand is warm and pink, with a capillary refill time of 2 seconds. Radiographs of the right elbow reveal a completely displaced extension-type supracondylar humerus fracture. Which of the following is the most appropriate next step in management?

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Answer: Closed reduction and percutaneous pinning

Answer

Closed reduction and percutaneous pinning
This patient presents with a completely displaced (Gartland type III) extension-type supracondylar humerus fracture accompanied by a classic 'pulseless, pink hand.' The inability to make an 'OK' sign indicates an anterior interosseous nerve (AIN) injury, which is the most commonly injured nerve in this specific fracture pattern. In a pulseless, pink hand, collateral circulation is sufficient to maintain viability, and the primary issue is mechanical kinking, tethering, or localized vasospasm of the brachial artery over the sharp proximal bone fragment. The standard of care is urgent closed reduction and percutaneous pinning (CRPP). Reducing the fracture restores anatomic alignment, which relieves the tension on the neurovascular bundle and typically restores the palpable pulse. Because the hand is well-perfused, there is no indication to delay skeletal stabilization for advanced imaging or vascular surgery.

Step-by-Step Solution

1
Identify the primary diagnosis and specific complications based on the clinical presentation.
The patient has a completely displaced (Gartland type III) extension-type supracondylar humerus fracture complicated by Anterior Interosseous Nerve (AIN) neuropraxia (inability to make the 'OK' sign) and a 'pulseless, pink hand'.
Accurate identification of the fracture pattern and neurovascular status dictates the treatment algorithm.
2
Analyze the vascular status of the extremity.
The radial pulse is absent (even on Doppler), but the hand remains warm, pink, and has normal capillary refill (< 2 seconds), indicating adequate collateral perfusion.
Differentiating between a 'pulseless, pink hand' (well-perfused) and a 'pulseless, pale hand' (ischemic) is critical for determining the urgency and type of intervention.
3
Select the most appropriate intervention according to standard pediatric orthopedic trauma algorithms.
The standard of care for a pulseless, pink hand in this setting is urgent closed reduction and percutaneous pinning (CRPP) without delaying for vascular studies.
The absent pulse is typically due to mechanical kinking or local spasm of the brachial artery over the proximal fracture fragment. Realignment of the bone reliably relieves this kinking and often restores the pulse.

Key Concept

Management of a pulseless but well-perfused (pink) hand in the setting of a pediatric displaced supracondylar humerus fracture.
Estimated Time:1m 30s
Question 69Question →

A 23-year-old male presents to the urgent care clinic with right wrist pain after falling on an outstretched hand while snowboarding. Physical examination reveals focal swelling and significant tenderness to palpation in the anatomic snuffbox. Initial anteroposterior, lateral, and scaphoid-view plain radiographs of the right wrist demonstrate no obvious fracture, dislocation, or joint effusion. Arrange the following management steps in the most appropriate chronological order for this patient.

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Answer

The correct sequence for managing a suspected scaphoid fracture with initially negative radiographs is to apply a thumb spica splint, re-evaluate the patient clinically in 10-14 days, obtain repeat plain radiographs at that follow-up, proceed with an MRI if radiographs remain negative but clinical suspicion is high, and finally refer to orthopedic surgery if a displaced fracture is confirmed.
Scaphoid fractures frequently do not appear on initial plain radiographs. Therefore, any patient presenting with a classic mechanism of injury (fall on an outstretched hand) and anatomic snuffbox tenderness must be treated empirically as having a scaphoid fracture until proven otherwise. The standard clinical algorithm dictates immediate immobilization in a thumb spica splint, followed by clinical re-evaluation and repeat radiographs in 10 to 14 days. During this interval, osteoclastic bone resorption at the fracture margins widens the fracture line, making an initially occult fracture visible on X-ray. If follow-up radiographs remain negative but the patient remains symptomatic, MRI is the most sensitive definitive diagnostic modality. Finally, if a displaced fracture or a fracture involving the proximal pole is identified, surgical fixation is required due to the high risk of avascular necrosis caused by the scaphoid's tenuous retrograde blood supply.

Step-by-Step Solution

1
Apply a thumb spica splint.
The wrist and thumb are fully immobilized.
Prevents displacement of a potential occult scaphoid fracture, significantly reducing the risk of avascular necrosis and nonunion.
2
Re-evaluate the patient in 10 to 14 days.
The patient returns for a follow-up assessment.
Provides sufficient time for initial acute swelling to subside and for natural osteoclastic activity to occur along any micro-fracture line.
3
Obtain repeat plain radiographs of the scaphoid.
Radiographic imaging is performed a second time.
Bone resorption at the fracture margins over the preceding 2 weeks often makes an initially occult fracture clearly visible on standard X-rays.
4
Order an MRI of the wrist.
High-resolution cross-sectional imaging is obtained.
Definitively rules in or rules out a scaphoid fracture if the repeat radiographs remain negative but the patient continues to exhibit snuffbox tenderness.
5
Refer to orthopedic surgery.
The patient is evaluated by a specialist for potential internal fixation.
Surgical intervention (e.g., screw fixation) is indicated if advanced imaging confirms a displaced fracture, which carries a high risk of avascular necrosis due to the scaphoid's retrograde blood supply.

Key Concept

Management algorithm for suspected scaphoid fractures with negative initial radiographs
Question 70Question →

A 34-year-old construction worker arrives at the trauma center following a workplace accident. He was struck on the right arm by a swinging metal pipe. He reports severe pain in his right arm. His vital signs are within normal limits. Examination of the right upper extremity reveals marked swelling and an obvious angular deformity in the middle of the upper arm. The overlying skin is intact. When asked to move his hand, the patient demonstrates a "wrist drop" and cannot actively straighten his fingers at the metacarpophalangeal joints. He reports numbness over the back of his right hand near the thumb. Radial and ulnar pulses are strong and symmetrical. An X-ray confirms a closed, displaced transverse fracture of the midshaft of the humerus. Which of the following is the most appropriate initial management for this patient?

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Answer: Placement of a coaptation splint and clinical observation

Answer

Placement of a coaptation splint and clinical observation
The correct initial management for a closed midshaft humerus fracture with a primary radial nerve palsy (present at the time of injury) is conservative, consisting of fracture stabilization (e.g., placement of a coaptation splint) and clinical observation. The radial nerve runs in the spiral groove along the posterior midshaft of the humerus, making it highly susceptible to injury. However, the majority of these injuries are neurapraxias (nerve contusion or stretching) rather than nerve transections, and they will recover spontaneously over 3 to 4 months. Immediate surgical exploration is not required unless the fracture is open, there is an associated vascular injury, or the nerve palsy develops only after an attempted closed reduction (suggesting nerve entrapment).

Step-by-Step Solution

1
Diagnose the primary injury and its complication from the clinical presentation and imaging.
The patient has a closed midshaft humerus fracture complicated by a primary radial nerve palsy (indicated by wrist drop and dorsal web space numbness).
Identifying the specific nerve involved and the nature of the fracture (closed vs. open) dictates the treatment algorithm.
2
Determine the indications for surgical intervention in midshaft humerus fractures.
Surgical indications include open fractures, vascular compromise, polytrauma, or a radial nerve palsy that develops after an attempted closed reduction. None of these are present.
Primary radial nerve palsy in a closed humerus fracture is most often a neurapraxia, which does not require acute surgical exploration.
3
Select the appropriate conservative management.
The correct management is immobilization of the fracture with a coaptation splint (or hanging arm cast) and close observation of the nerve deficit.
Fracture stabilization promotes healing, and allowing time for spontaneous nerve recovery avoids unnecessary surgical risks.

Key Concept

Management of closed midshaft humerus fractures with primary radial nerve palsy
Estimated Time:1m 30s
Question 71Question →

Match each primary bone tumor with its most characteristic radiographic finding and classic anatomical location.

Click a left item, then click its matching right item

Items

Osteosarcoma
Ewing sarcoma
Giant cell tumor of bone
Osteoid osteoma

Matches

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Answer

Osteosarcoma matches with the metaphyseal lesion showing a sunburst pattern. Ewing sarcoma matches with the diaphyseal lesion showing an onion-skin pattern. Giant cell tumor matches with the epiphyseal lytic soap-bubble lesion. Osteoid osteoma matches with the radiolucent nidus surrounded by thick sclerosis.
Each primary bone tumor has a classic pathognomonic radiographic appearance and predilection for a specific segment of the long bone (epiphysis, metaphysis, or diaphysis). Osteosarcoma favors the metaphysis (sunburst), Ewing sarcoma favors the diaphysis (onion-skin), Giant cell tumor favors the epiphysis (soap-bubble), and Osteoid osteoma produces a characteristic sclerotic nidus.

Step-by-Step Solution

1
Analyze the location and radiographic pattern associated with Osteosarcoma.
Identified as a metaphyseal lesion with a 'sunburst' periosteal reaction and Codman triangle.
Osteosarcoma is a highly malignant primary bone tumor that classically occurs at the metaphysis of long bones and induces aggressive, disorganized new bone formation.
2
Analyze the location and radiographic pattern associated with Ewing sarcoma.
Identified as a diaphyseal permeative lesion with an 'onion-skin' periosteal reaction.
Ewing sarcoma stems from neuroectodermal cells, typically growing in the marrow cavity of the diaphysis and causing concentric layers of reactive bone.
3
Analyze the location and radiographic pattern associated with Giant cell tumor of bone.
Identified as an epiphyseal, eccentric lytic lesion with a 'soap bubble' appearance.
Giant cell tumors are locally aggressive but generally benign lesions that classically affect the epiphyseal region of closed growth plates.
4
Analyze the radiographic pattern associated with Osteoid osteoma.
Identified as a small radiolucent cortical nidus surrounded by dense sclerosis.
Osteoid osteomas are small benign tumors where the prostaglandin-secreting central nidus triggers prominent surrounding reactive bone formation.

Key Concept

Radiographic and anatomical characteristics of primary bone tumors.
Estimated Time:1m 30s
Question 72Question →

A 38-year-old man is brought to the emergency department after being struck by a motor vehicle while crossing the street. On arrival, he is confused but protecting his airway. His temperature is 36.5∘C36.5^\circ\text{C}, blood pressure is 78/40 mmHg78/40\text{ mmHg}, pulse is 135/min135/\text{min}, and respiratory rate is 26/min26/\text{min}. Oxygen saturation is 96%96\% on room air. Primary survey reveals equal and bilateral breath sounds. The abdomen is soft and non-distended. Examination of the pelvis reveals gross instability to anteroposterior compression. There is a large scrotal hematoma and a prominent drop of blood at the urethral meatus. An extended focused assessment with sonography for trauma (E-FAST) reveals no free fluid in the pericardial, pleural, or peritoneal spaces. A pelvic binder is immediately applied, and the patient is resuscitated with 2 liters of isotonic crystalloid and 2 units of uncrossmatched type O-negative packed red blood cells. Re-evaluation 15 minutes later shows a blood pressure of 82/45 mmHg82/45\text{ mmHg} and a pulse of 130/min130/\text{min}. Which of the following is the most appropriate next step in management?

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Answer: Pelvic angiography with embolization

Answer

Pelvic angiography with embolization is the correct immediate intervention to control ongoing retroperitoneal hemorrhage in a hemodynamically unstable patient with a pelvic fracture and a negative E-FAST.
The correct answer addresses the immediate life-threatening issue: ongoing hemorrhage from a pelvic fracture. The patient remains in shock despite appropriate initial interventions (pelvic binder and blood products). Because the E-FAST is negative, the hemorrhage is isolated to the retroperitoneum (a space not adequately visualized by FAST). The standard algorithm for an unstable pelvic fracture with a negative E-FAST is to proceed directly to pelvic angiography with embolization (or preperitoneal pelvic packing) to arrest the bleeding.

Step-by-Step Solution

1
Assess the patient's hemodynamic status and identify life-threatening injuries.
The patient is in hemorrhagic shock (hypotension, tachycardia) and has a mechanically unstable pelvic fracture, which is a major source of blood loss.
Prioritizing ABCs and hemorrhage control is the foundation of trauma management.
2
Evaluate the results of the E-FAST exam and the initial resuscitative efforts.
The E-FAST is negative (ruling out massive intraperitoneal or intrathoracic bleeding), and the patient remains hemodynamically unstable despite a pelvic binder and blood transfusions.
Determining the source of bleeding dictates the surgical or endovascular approach.
3
Select the definitive intervention for ongoing pelvic hemorrhage.
Pelvic angiography with embolization (or preperitoneal pelvic packing) is chosen.
Since the bleeding is localized to the retroperitoneal space, angiographic embolization directly targets the bleeding pelvic vessels without opening the abdomen and disrupting the natural tamponade effect.

Key Concept

Management algorithm for hemodynamically unstable pelvic fractures
Estimated Time:2m 0s
Question 73Question →

An 8282-year-old woman is brought to the emergency department after a mechanical fall at home. She tripped over a rug and landed directly on her right side. She complains of severe right groin pain and is completely unable to bear weight. Her past medical history is significant for osteoporosis, hypertension, and mild chronic obstructive pulmonary disease. Her current medications include amlodipine, alendronate, and an albuterol inhaler.

Vital signs are temperature 37.1∘C37.1^\circ\text{C} (98.8∘F98.8^\circ\text{F}), blood pressure 138/82 mm Hg138/82\text{ mm Hg}, pulse 88/min88/\text{min}, and respiratory rate 16/min16/\text{min}. On physical examination, her right lower extremity is visibly shortened and externally rotated. Palpation over the greater trochanter elicits significant tenderness. Distal pulses are intact, and there are no sensory deficits.

Anteroposterior radiographs of the right hip reveal a completely displaced fracture line extending transversely across the anatomical neck of the right femur, entirely proximal to the intertrochanteric line. There is no evidence of underlying osteolytic lesions.

Which of the following is the most appropriate definitive management for this patient's injury?

Show answer & explanation

Answer: Hemiarthroplasty of the right hip

Answer

Hemiarthroplasty of the right hip
The correct management for a displaced, intracapsular femoral neck fracture in an elderly patient is a hemiarthroplasty (or total hip arthroplasty). The femoral head receives its primary blood supply from the medial circumflex femoral artery, which travels in a retrograde fashion along the femoral neck. A completely displaced fracture of the anatomical neck tears these vessels, placing the patient at an extremely high risk for avascular necrosis and nonunion. Because head-preserving surgeries (like percutaneous pinning) have unacceptably high failure rates in this specific demographic and fracture pattern, prosthetic replacement is required to allow for immediate weight-bearing and early mobilization.

Step-by-Step Solution

1
Analyze the clinical presentation and physical exam.
The patient is an elderly woman with a shortened, externally rotated right leg following a fall, which is the classic presentation for a hip fracture.
Identifying the primary injury is the first step in orthopedic trauma assessment.
2
Interpret the specific radiographic findings.
The fracture is described as completely displaced and extending across the anatomical neck, which classifies it as an intracapsular femoral neck fracture.
Precise anatomical classification (intracapsular vs. extracapsular) dictates the surgical approach because of differences in blood supply to the femoral head.
3
Select the appropriate definitive intervention.
Due to the high risk of avascular necrosis from disruption of the medial circumflex femoral artery in displaced intracapsular fractures, joint replacement (hemiarthroplasty) is indicated over internal fixation.
Head-preserving surgeries have unacceptably high failure rates in this specific demographic and fracture pattern.

Key Concept

Displaced intracapsular femoral neck fractures in the elderly carry a high risk of avascular necrosis and are optimally treated with arthroplasty (e.g., hemiarthroplasty) rather than internal fixation.

Alternative Method

While a total hip arthroplasty (THA) is an alternative to hemiarthroplasty for displaced femoral neck fractures, THA is typically reserved for younger, higher-demand patients or those with pre-existing severe osteoarthritis of the acetabulum. For an 82-year-old with lower baseline activity, hemiarthroplasty provides excellent pain relief with a lower dislocation risk and a shorter overall surgical time.
Estimated Time:1m 15s
Question 74Question →

A young adult is transported to the trauma center via helicopter after a high-speed motorcycle crash. Following completion of the primary trauma survey and hemodynamic stabilization, evaluation reveals an isolated, severe left lower extremity injury. Examination shows a highly comminuted, open mid-shaft tibia fracture with a 12-cm soft tissue defect, extensive contamination, and exposed bone. The ipsilateral foot is pale and cold. Doppler ultrasonography demonstrates absent signals over the dorsalis pedis and posterior tibial arteries. Immediate operative intervention is planned. Arrange the following surgical management steps in the most appropriate chronological order for this patient.

Drag items to arrange them in the correct order

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Answer

The correct chronological sequence is: 1) Aggressive surgical debridement and irrigation, 2) Application of a temporary external skeletal fixator, 3) Definitive arterial repair, 4) Prophylactic four-compartment fasciotomy, and 5) Delayed definitive soft tissue coverage.
The standard of care for a Gustilo Type IIIC open fracture mandates initial aggressive debridement to control contamination. This is immediately followed by skeletal stabilization (typically external fixation) to establish a rigid framework. Only after the bone is stable should definitive vascular repair be performed, as manipulating unstable bone ends would jeopardize a newly completed vascular anastomosis. Reperfusion of an ischemic limb reliably causes significant tissue edema, necessitating prophylactic fasciotomies during the index operation. Finally, definitive soft tissue coverage is delayed for several days until the wound bed is pristine and the patient is physiologically optimized.

Step-by-Step Solution

1
Identify the injury severity and classification
The scenario describes a Gustilo-Anderson Type IIIC open tibia fracture.
Characterized by a severe soft tissue defect, extensive contamination, bone exposure, and an associated arterial injury requiring repair.
2
Determine the first intraoperative priority
Aggressive surgical debridement and irrigation.
Removing necrotic tissue and contamination is the cornerstone of open fracture management and must occur before any hardware or grafts are placed.
3
Determine the sequence of skeletal vs. vascular reconstruction
Skeletal stabilization (external fixation) is performed before definitive vascular repair.
Fixing the bone first provides a stable framework, ensuring that the traction and manipulation required for orthopedic reduction do not tear the delicate vascular anastomosis.
4
Address the physiologic consequences of reperfusion
Perform prophylactic four-compartment fasciotomies.
Restoring blood flow to an ischemic limb causes massive capillary leak and edema (ischemia-reperfusion injury), making acute compartment syndrome highly likely if fasciae are left intact.
5
Plan for final wound management
Delay definitive soft tissue coverage (free flap).
Immediate closure or coverage of a highly contaminated wound risks deep infection; it is deferred until subsequent debridements yield a pristine wound bed.

Key Concept

Surgical Sequencing for Gustilo Type IIIC Open Fractures (Mangled Extremity)

Alternative Method

If the warm ischemia time exceeds 6 hours, the standard sequence is modified: a temporary intravascular shunt (e.g., Argyle shunt) is placed FIRST to immediately restore distal perfusion. This is followed by external fixation, definitive vascular repair (replacing the shunt), and fasciotomies.
Estimated Time:1m 30s
Question 75Question →

A 22-year-old man is evaluated in the hospital for severe right leg pain. Ten hours ago, he underwent closed reduction and placement of a circumferential long-leg cast for a closed, displaced midshaft tibia fracture sustained during a soccer match. He states that the pain is agonizing, describes it as a deep 'burning' sensation, and reports that it has progressively worsened over the last three hours despite receiving scheduled doses of intravenous hydromorphone. On physical examination, the right foot is warm and pink. Capillary refill is 2 seconds, and the dorsalis pedis and posterior tibial pulses are palpable. He holds his ankle in slight plantar flexion. Passive dorsiflexion of the right toes elicits excruciating pain. Sensation to light touch is decreased over the first dorsal web space. Which of the following is the most appropriate initial step in the management of this patient?

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Answer: Immediate bivalving of the cast and division of underlying padding

Answer

Immediate bivalving of the cast and division of underlying padding
The correct answer is the immediate bivalving of the cast and division of underlying padding. This patient exhibits classic early signs of acute compartment syndrome (ACS), particularly in the anterior compartment of the lower leg. The anterior compartment contains the muscles that dorsiflex the foot and the deep peroneal nerve, which provides sensation to the first dorsal web space. Severe pain out of proportion to the injury, intense pain on passive stretch (dorsiflexion), and early sensory changes are highly specific for ACS. In any patient with suspected ACS who has a cast or circumferential dressing, the immediate first step is to relieve all external compression. Bivalving the cast and cutting all underlying cotton padding down to the skin can drastically reduce compartment pressure and may restore adequate perfusion. The limb should also be kept at the level of the heart to maximize the arterial pressure gradient.

Step-by-Step Solution

1
Identify the clinical syndrome based on the presentation.
The patient has pain out of proportion to the injury, pain with passive stretching of the muscles (dorsiflexion), and early neurologic deficits (decreased sensation in the first web space), indicating Acute Compartment Syndrome (ACS).
These are the classic early signs of ACS. The deep peroneal nerve runs through the anterior compartment and supplies sensation to the first dorsal web space.
2
Determine the most appropriate immediate intervention.
The initial step is to completely remove any external constricting forces.
Bivalving the cast and cutting all underlying padding down to the skin can reduce intracompartmental pressure by 50% to 85%, potentially averting the need for surgical fasciotomy.

Key Concept

The hallmark early signs of acute compartment syndrome are pain out of proportion to the injury and pain on passive stretch; the immediate first step in a patient with a circumferential dressing or cast is bivalving the cast and dividing all padding to the skin.
Question 76Question →

A 32-year-old man is evaluated in the orthopedic ward for severe left leg pain. Twelve hours ago, he underwent closed reduction and placement of a long-leg cast for a closed midshaft tibial fracture. He now reports excruciating, throbbing pain in his calf that is worsening and no longer relieved by intravenous morphine. Physical examination reveals a tense, 'tight' feeling in the calf musculature and profound pain with passive dorsiflexion of the toes. He also reports new-onset tingling in the first web space of his foot. Distal pulses remain palpable. Arrange the following steps in the most appropriate chronological order for the acute evaluation and management of this patient's condition.

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Answer

The correct sequence is: 1) Bivalve the cast and release dressings, 2) Position the extremity at heart level, 3) Measure intra-compartmental pressures, and 4) Perform an emergent fasciotomy.
Acute compartment syndrome is a limb-threatening emergency. The management protocol follows a strict hierarchy: first, remove any external compressive forces (such as a cast or tight dressing) which can immediately drop pressures. Second, optimize hemodynamics by placing the limb level with the heart. Third, if the diagnosis remains ambiguous or symptoms fail to improve, objectively measure compartment pressures. Finally, once confirmed, proceed immediately to the operating room for definitive surgical decompression via fasciotomy.

Step-by-Step Solution

1
Recognize the signs of acute compartment syndrome (ACS).
The patient exhibits pain out of proportion, pain with passive stretch, and paresthesias.
Early recognition is crucial as ACS is a time-sensitive surgical emergency.
2
Determine the first immediate intervention.
The initial action is to bivalve the cast and release all circumferential padding.
External compression is a major reversible cause of elevated compartment pressures.
3
Determine the optimal limb positioning.
The limb must be positioned at the level of the heart.
Elevation decreases arterial perfusion pressure, which worsens the ischemic insult.
4
Decide on the next diagnostic step if symptoms do not rapidly resolve.
Measure intra-compartmental pressures using a manometer.
Objective confirmation is needed before proceeding to highly morbid surgical decompression.
5
Identify the definitive treatment.
Perform an emergent four-compartment fasciotomy.
Surgical release of the fascia is the only definitive way to restore tissue perfusion once ACS is established.

Key Concept

Evaluation and management sequence for acute compartment syndrome
Question 77Question →

Match each patient's clinical presentation of an orthopedic injury with the most likely associated neurovascular complication or mandatory physical examination finding.

Click a left item, then click its matching right item

Items

A 24-year-old man with a deformed arm after a direct blow; radiographs show a transverse fracture at the middle third of the humerus.
A 19-year-old man holding his right arm in slight abduction and external rotation after a fall; the acromion is prominent.
A 32-year-old woman in a high-speed collision presenting with a shortened, internally rotated, and adducted right lower extremity.
A 28-year-old pedestrian struck by a car presenting with gross multidirectional instability of the tibiofemoral joint.
A 7-year-old boy with a swollen, painful elbow after a fall; radiographs show an extension-type fracture just above the condyles.

Matches

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Answer

Midshaft humerus fracture matches with inability to extend wrist. Anterior shoulder dislocation matches with decreased lateral shoulder sensation. Posterior hip dislocation matches with weakness of ankle dorsiflexion. Knee dislocation matches with diminished distal pulses requiring ABI. Pediatric supracondylar fracture matches with inability to flex distal index finger and thumb.
Correct matching relies on understanding the anatomical relationships between bones, joints, and adjacent neurovascular structures. Midshaft humerus fractures threaten the radial nerve; anterior shoulder dislocations threaten the axillary nerve; posterior hip dislocations threaten the sciatic nerve; knee dislocations carry a high risk of popliteal artery injury; and pediatric supracondylar humerus fractures commonly injure the anterior interosseous branch of the median nerve.

Step-by-Step Solution

1
Analyze the presentation of the midshaft humerus fracture.
Identified risk to the radial nerve.
The radial nerve courses through the spiral groove of the humerus, making it highly susceptible to injury in midshaft fractures, leading to impaired wrist and finger extension.
2
Analyze the presentation of the anterior shoulder dislocation.
Identified risk to the axillary nerve.
The axillary nerve wraps around the surgical neck of the humerus and can be stretched during an anterior dislocation, causing loss of sensation over the lateral deltoid.
3
Analyze the presentation of the shortened, internally rotated, and adducted lower extremity.
Diagnosed as a posterior hip dislocation, identifying risk to the sciatic nerve.
The sciatic nerve runs directly posterior to the hip joint. A posterior dislocation can compress it, most commonly affecting the common peroneal division and causing foot drop.
4
Analyze the presentation of the tibiofemoral joint instability.
Diagnosed as a knee dislocation, identifying critical risk to the popliteal artery.
The popliteal artery is rigidly tethered proximally and distally to the knee joint, making it highly vulnerable to tethering and intimal tearing during a gross dislocation.
5
Analyze the presentation of the pediatric extension-type supracondylar humerus fracture.
Identified risk to the anterior interosseous nerve.
The anterior interosseous nerve (a branch of the median nerve) is the most commonly injured nerve in extension-type supracondylar fractures, leading to weakness of the flexor pollicis longus and flexor digitorum profundus (loss of the 'OK' sign).

Key Concept

Neurovascular complications of specific orthopedic injuries
Question 78Question →

A 13-year-old boy is brought to the clinic by his parents due to left knee pain and a limp that has progressively worsened over the past month. Yesterday, the pain became significantly more severe after he jumped down from a small wall. He has no history of direct trauma to the knee, fever, or prior medical conditions. His BMI is 34 kg/m234 \text{ kg/m}^2. Vital signs are within normal limits. Physical examination of the left knee reveals no effusion, joint line tenderness, or ligamentous laxity. During examination of the left hip, flexion of the hip results in obligatory external rotation of the thigh. Internal rotation of the left hip is severely limited and elicits pain. Radiographs of the hip confirm the diagnosis. Which of the following is the most appropriate next step in management?

Show answer & explanation

Answer: Strict non-weight bearing and operative stabilization

Answer

Strict non-weight bearing and operative stabilization
The patient's presentation of a limp, referred knee pain, obesity, and obligatory external rotation of the thigh during hip flexion is highly characteristic of a slipped capital femoral epiphysis (SCFE). In SCFE, the proximal femoral epiphysis slips posteriorly and inferiorly relative to the femoral neck. Because the condition can rapidly progress and lead to avascular necrosis of the femoral head, the immediate next step in management is to ensure the patient is strictly non-weight bearing and to consult orthopedics for surgical stabilization (typically percutaneous pinning in situ).

Step-by-Step Solution

1
Analyze the physical examination findings to localize the source of the pain.
The knee exam is entirely normal, while the hip exam shows limited internal rotation and obligatory external rotation upon flexion.
Knee pain in an adolescent can often be referred from the hip due to shared innervation by the obturator nerve. A normal knee exam necessitates a careful hip evaluation.
2
Integrate the demographic data and localized hip pathology to form a diagnosis.
An obese 13-year-old male with chronic hip/referred knee pain and altered hip mechanics has Slipped Capital Femoral Epiphysis (SCFE).
SCFE classically presents in overweight adolescents undergoing rapid growth spurts, resulting in the proximal femoral epiphysis slipping posteriorly and inferiorly.
3
Determine the definitive management for the diagnosed condition.
The most appropriate next step is strict non-weight bearing and surgical fixation.
SCFE is an orthopedic emergency because further slippage can compromise the blood supply to the femoral head, leading to avascular necrosis. Immediate surgical pinning in situ is required.

Key Concept

Diagnosis and management of Slipped Capital Femoral Epiphysis (SCFE)
Estimated Time:1m 15s
Question 79Question →

Correlate each pediatric or adolescent patient's musculoskeletal presentation with the most accurate orthopedic diagnosis.

Click a left item, then click its matching right item

Items

A 6-year-old male is evaluated for a progressive, painless gait abnormality and notably decreased internal rotation of the right hip over a four-month period.
A 12-year-old adolescent with a body mass index in the 98th percentile reports sudden-onset groin discomfort and rests the affected leg in a continuously externally rotated position.
A 5-year-old girl is unwilling to walk on her right leg a week after recovering from rhinorrhea and a sore throat; her vital signs and serum inflammatory markers are strictly within normal limits.
A 14-year-old athlete describes persistent, localized pain and a palpable bony prominence just inferior to the patella, which becomes severely irritated during basketball practice.

Matches

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Answer

The correct pairings identify Legg-Calvé-Perthes disease, slipped capital femoral epiphysis, transient synovitis, and Osgood-Schlatter disease based on their unique epidemiological profiles and physical exam findings.
Each diagnosis is correctly matched by synthesizing the specific epidemiological data (such as age and weight) with clinical signs (such as recent illness, location of the pain, and specific range of motion limitations) unique to each pediatric musculoskeletal pathology.

Step-by-Step Solution

1
Analyze the 6-year-old boy with a chronic, painless limp.
Identify Legg-Calvé-Perthes disease.
The insidious onset in early school-aged boys combined with limited internal rotation suggests avascular necrosis of the proximal femoral epiphysis.
2
Evaluate the obese 12-year-old with acute groin pain and external rotation.
Identify slipped capital femoral epiphysis.
The patient's elevated body mass index and the characteristic resting position of external rotation point to posterior displacement of the femoral epiphysis.
3
Assess the 5-year-old girl refusing to bear weight after a viral illness.
Identify transient synovitis.
The recent history of an upper respiratory infection and the absence of systemic inflammation (normal vitals/labs) rule out septic arthritis, making transient synovitis the most likely diagnosis.
4
Review the 14-year-old athlete with pain at the tibial tubercle.
Identify Osgood-Schlatter disease.
Activity-related pain localized inferior to the patella at the tibial tuberosity in an active adolescent is a classic description of this traction injury.

Key Concept

Accurate differentiation of pediatric orthopedic conditions relies heavily on patient age, body habitus, acuity of onset, preceding illnesses, and precise anatomical localization of pain.
Question 80Question →

A 25-year-old man is brought to the emergency department after a skiing accident in which he fell directly onto his right shoulder. He complains of severe shoulder pain and an inability to move his arm. Vital signs are within normal limits. On physical examination, the right arm is held in slight abduction and external rotation. There is a prominent acromion and a loss of the normal rounded contour of the shoulder. He has decreased sensation to pinprick over the lateral aspect of the right shoulder. Radial and brachial pulses are 2+ and symmetric. Capillary refill is less than 2 seconds. Anteroposterior and axillary lateral radiographs of the right shoulder demonstrate an anterior, subcoracoid dislocation of the humeral head. The patient undergoes closed reduction in the emergency department using procedural sedation. Post-reduction radiographs confirm anatomic alignment of the glenohumeral joint. However, upon repeat physical examination, the patient is able to actively abduct the arm to 15 degrees, but is unable to actively abduct it any further. The decreased sensation over the lateral shoulder persists. Which of the following is the most appropriate next step in the management of this patient?

Show answer & explanation

Answer: Application of a shoulder sling and outpatient physical therapy

Answer

Application of a shoulder sling and outpatient physical therapy
The patient's ability to initiate abduction to 15 degrees confirms that the supraspinatus muscle is intact. However, his inability to abduct past 15 degrees isolates a functional deficit in the deltoid muscle. Combined with the sensory loss over the lateral shoulder (the 'regimental badge' area), this is the classic presentation of an axillary nerve injury. The axillary nerve is frequently stretched over the humeral head during an anterior shoulder dislocation. Because the vast majority of these injuries are neuropraxias that recover spontaneously within 6 to 12 weeks, the standard of care is conservative management. Applying a shoulder sling for comfort and initiating outpatient physical therapy maintains joint mobility while awaiting nerve recovery.

Step-by-Step Solution

1
Identify the primary injury from the initial presentation.
The mechanism, arm position, and loss of contour indicate an anterior shoulder dislocation, which is confirmed by the radiographs.
Establishing the primary trauma guides the assessment of specific associated complications.
2
Analyze the post-reduction physical examination findings.
The patient can initiate abduction (0-15 degrees) but cannot continue it, and has sensory loss over the lateral shoulder.
Identifying the specific muscle weakness isolates the affected anatomical structure.
3
Correlate the physical findings with functional anatomy.
The supraspinatus (suprascapular nerve) handles 0-15 degrees of abduction, while the deltoid (axillary nerve) handles 15-90 degrees. The sensory loss is in the regimental badge area.
This confirms an isolated axillary nerve injury, sparing the suprascapular nerve and rotator cuff.
4
Determine the standard of care for this specific complication.
Axillary nerve injuries from anterior dislocations are typically traction neuropraxias that resolve spontaneously over weeks to months.
Understanding the natural history prevents unnecessary invasive interventions or premature diagnostic testing.

Key Concept

Axillary nerve injury is a common complication of anterior shoulder dislocations, presenting with deltoid weakness (impaired abduction beyond 15 degrees) and lateral shoulder numbness; it is initially managed conservatively with a sling and physical therapy.

Alternative Method

When evaluating shoulder weakness after a dislocation, always separate the phases of abduction: 0-15 degrees tests the suprascapular nerve (supraspinatus), and 15-90 degrees tests the axillary nerve (deltoid).
Estimated Time:1m 30s
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