Orthopedic Surgery

132 questions

Question 1Question →

A 45-year-old man is brought to the emergency department via ambulance after falling approximately 20 feet from a roof. Upon arrival, he is moaning in pain and appears confused. His temperature is 36.1°C (97.0°F), blood pressure is 78/40 mmHg, pulse is 135/min, and respirations are 26/min. The airway is patent, and breath sounds are equal bilaterally. Physical examination reveals severe ecchymosis over the lower abdomen and perineum. The pelvis is mechanically unstable to gentle anteroposterior compression. The right lower extremity is shortened and externally rotated. A focused assessment with sonography for trauma (FAST) examination reveals no intraperitoneal or pericardial fluid. An anteroposterior radiograph of the pelvis demonstrates a 4-cm diastasis of the pubic symphysis and widening of the right sacroiliac joint. A pelvic binder is immediately applied, and a massive transfusion protocol is initiated. After receiving 2 units of uncrossmatched packed red blood cells and 1 unit of fresh frozen plasma, his blood pressure remains 80/42 mmHg and pulse is 130/min. Which of the following is the most appropriate next step in management?

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Answer: Pelvic angiography with embolization

Answer

The most appropriate next step in management is pelvic angiography with embolization.
The patient presents with an open-book pelvic fracture and hemorrhagic shock. Initial management correctly included the application of a pelvic binder, which reduces pelvic volume and helps tamponade venous bleeding, alongside aggressive resuscitation. However, he remains hemodynamically unstable despite these measures. A negative FAST examination indicates there is no major intraperitoneal hemorrhage, confirming that the ongoing bleeding is localized to the retroperitoneal space. The most appropriate next step to achieve hemostasis in this scenario is pelvic angiography with embolization to control arterial bleeding (typically from branches of the internal iliac artery). Preperitoneal pelvic packing is an alternative definitive intervention.

Step-by-Step Solution

1
Assess the patient's hemodynamic status and trauma primary survey.
The patient is in hemorrhagic shock (blood pressure 80/42 mmHg) and is not responding adequately to mechanical stabilization and massive transfusion.
To determine the acuity and prioritize life-saving interventions over secondary diagnostic adjuncts.
2
Interpret the results of the FAST examination and pelvic radiograph.
The FAST is negative, ruling out massive intraperitoneal hemorrhage. The radiograph confirms an unstable open-book pelvic fracture.
To localize the source of shock, which in this case is retroperitoneal bleeding originating from the disrupted pelvic ring.
3
Determine the algorithmic next step for refractory pelvic bleeding.
Because the bleeding is retroperitoneal and unresponsive to mechanical stabilization (pelvic binder), arterial hemostasis is required.
Venous bleeding is often controlled by the binder; persistent shock implies arterial injury requiring angiography or preperitoneal packing.

Key Concept

In a hemodynamically unstable patient with a pelvic fracture and a negative FAST examination, the most appropriate next step after placing a pelvic binder and initiating resuscitation is pelvic angiography with embolization to control retroperitoneal arterial hemorrhage.
Question 2Question →

A series of patients present to the trauma bay with various acute orthopedic injuries. Match each specific fracture pattern or dislocation with its most likely associated neurovascular deficit observed on physical examination.

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Items

Anterior shoulder dislocation
Midshaft humeral fracture
Supracondylar humeral fracture with posterolateral displacement
Medial humeral epicondyle fracture
Fibular neck fracture

Matches

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Answer

Anterior shoulder dislocation pairs with axillary nerve deficits; midshaft humeral fracture pairs with radial nerve deficits; posterolateral supracondylar fracture pairs with median/anterior interosseous nerve deficits; medial epicondyle fracture pairs with ulnar nerve deficits; and fibular neck fracture pairs with common peroneal nerve deficits.
Each match correctly pairs a classic orthopedic injury with the peripheral nerve most vulnerable at that specific anatomical site, accurately describing the resulting motor and sensory deficits.

Step-by-Step Solution

1
Analyze each specific orthopedic fracture or dislocation and identify the major peripheral nerve running in closest anatomical proximity to the injury site.
Identified Axillary, Radial, Median (AIN), Ulnar, and Common Peroneal nerves respectively.
Anatomical proximity is the primary determinant of which neurovascular structure is at risk during a fracture or dislocation.
2
Determine the motor and sensory distribution of each identified nerve.
Correlated the nerves with their respective physical exam findings, such as wrist drop for the radial nerve and loss of the 'OK' sign for the anterior interosseous nerve.
Clinical presentation of nerve injury depends strictly on the distal innervation pattern of the compromised nerve.
3
Match the orthopedic injury to the corresponding clinical deficit.
All five pairs are successfully matched based on neuroanatomical principles.
Accurate pairing ensures correct diagnosis and timely intervention for traumatic neuropathies.

Key Concept

Neurovascular complications of orthopedic trauma based on anatomical proximity.
Question 3Question →

Match each upper extremity fracture or dislocation pattern with its most classically associated neurovascular complication.

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Items

Lunate dislocation
Hook of hamate fracture
Monteggia fracture-dislocation
Scaphoid waist fracture

Matches

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Answer

Lunate dislocation pairs with acute median nerve compression; Hook of hamate fracture pairs with ulnar nerve compression; Monteggia fracture-dislocation pairs with posterior interosseous nerve injury; Scaphoid waist fracture pairs with interruption of retrograde blood supply.
Each pair correctly matches an upper extremity injury to its anatomical neurovascular consequence. Anterior dislocation of the lunate pushes the bone into the carpal tunnel, compressing the median nerve. The hook of the hamate sits adjacent to Guyon's canal, making fractures there a prime cause of ulnar nerve palsy. Monteggia fractures result in radial head dislocation, which stretches the posterior interosseous nerve (PIN) as it pierces the supinator muscle, leading to isolated motor deficits. Finally, the scaphoid is supplied by blood vessels that enter distally and travel proximally; a fracture at the waist interrupts this flow, causing avascular necrosis of the proximal fragment.

Step-by-Step Solution

1
Identify the anatomical location and structures involved in each specific upper extremity injury.
Lunate (carpal tunnel), hook of hamate (Guyon's canal), Monteggia (proximal radioulnar joint), scaphoid (distal-to-proximal vascular supply).
Understanding local anatomy is essential for predicting which nerves or blood vessels are at risk during displacement or swelling.
2
Correlate the displaced bone or fracture line with the adjacent neurovascular structures.
Dislocated lunate compresses the median nerve. Hamate fractures compress the ulnar nerve. Radial head dislocation (Monteggia) stretches the posterior interosseous nerve. Scaphoid fractures disrupt retrograde arterial flow.
The physical proximity of the injury directly dictates the resulting complication.

Key Concept

Neurovascular complications of forearm and wrist injuries
Question 4Question →

During a collegiate rugby match, an 18-year-old athlete sustains a high-impact direct blow to the anterior right proximal tibia while the foot is firmly planted on the turf. The athlete falls to the ground in severe distress and is unable to bear weight. On examination in the emergency department 45 minutes later, there is a prominent step-off at the right knee joint, with the tibia displaced posteriorly relative to the femur. The overlying skin is intact. The patient reports severe pain but denies any numbness or tingling in the foot. Dorsalis pedis and posterior tibial pulses are palpable and symmetric to the uninjured leg. Capillary refill is less than 2 seconds. A bedside closed reduction is performed promptly under conscious sedation, successfully restoring normal anatomic alignment of the knee. Post-reduction, the distal pulses remain palpable and symmetric. Which of the following is the most appropriate next step in the management of this patient?

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Answer: Measure the ankle-brachial index

Answer

Measure the ankle-brachial index
Knee dislocations involve extreme displacement of the tibia relative to the femur, which exerts tremendous sheer and traction forces on the tethered popliteal artery. Because up to one-third of knee dislocations are associated with popliteal artery injury, ruling out vascular compromise is the most critical next step. The presence of palpable distal pulses and brisk capillary refill are reassuring 'soft signs,' but they do not definitively exclude an intimal tear, which can progress to complete thrombosis and limb ischemia. Therefore, the standard of care is to measure the ankle-brachial index (ABI) in all patients with a knee dislocation and normal pulses. If the ABI≥0.9ABI \geq 0.9, the patient can be managed with serial neurovascular checks. If the ABI<0.9ABI < 0.9, or if distal pulses are asymmetric, computed tomography (CT) angiography is required.

Step-by-Step Solution

1
Identify the injury based on the mechanism and physical exam findings.
The patient suffered a posterior knee dislocation, which was successfully reduced.
Recognizing the injury pattern is crucial because knee dislocations are high-energy injuries tightly associated with specific neurovascular complications.
2
Assess the risk of associated complications.
The popliteal artery is firmly anchored proximally and distally to the knee joint, making it highly susceptible to stretch, intimal tear, or complete transection during a dislocation.
Vascular compromise is the most limb-threatening complication of a knee dislocation.
3
Determine the appropriate diagnostic algorithm for a patient with palpable pulses post-reduction.
Even with palpable pulses (which can be maintained by collateral circulation or partial tears), an ankle-brachial index (ABI) must be measured.
An ABI ABI≥0.9ABI \geq 0.9 can safely allow for serial observation, while an ABI<0.9ABI < 0.9 mandates computed tomography (CT) angiography to locate the vascular injury.

Key Concept

All knee dislocations require mandatory vascular assessment with an ankle-brachial index (ABI), even if distal pulses are palpable, to rule out occult popliteal artery injury.
Estimated Time:1m 0s
Question 5Question →

A 45-year-old man is brought to the emergency department after a heavy concrete pipe rolled over his lower torso at a construction site. On arrival, his airway is patent, and breath sounds are equal bilaterally. His blood pressure is 78/45 mmHg and his pulse is 132/min. There is no visible external hemorrhage. Physical examination reveals ecchymosis over the perineum and severe instability of the pelvic ring upon gentle lateral compression. A focused assessment with sonography for trauma (FAST) examination reveals no intraperitoneal free fluid. Which of the following is the most appropriate next step in management?

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Answer: Application of a circumferential pelvic binder

Answer

Application of a circumferential pelvic binder
The correct answer is the application of a circumferential pelvic binder. This patient presents with class III/IV hemorrhagic shock (hypotension, marked tachycardia) and signs of an unstable pelvic ring fracture following a crush injury. In the setting of blunt trauma, a negative focused assessment with sonography for trauma (FAST) examination indicates that there is no significant intraperitoneal free fluid. Therefore, the source of bleeding is most likely retroperitoneal, arising directly from the fractured cancellous bone and the disrupted presacral venous plexus. The immediate priority in managing a hemodynamically unstable patient with a pelvic fracture is mechanical stabilization. A circumferential pelvic binder (or a firmly wrapped sheet centered over the greater trochanters) reduces the internal volume of the pelvis. This volume reduction promotes the tamponade of venous bleeding and prevents the disruption of early hemostatic clots by limiting bone motion, effectively addressing the 'Circulation' component of the Advanced Trauma Life Support (ATLS) protocol.

Step-by-Step Solution

1
Assess the patient's hemodynamic status.
Blood pressure is 78/45 mmHg and pulse is 132/min, indicating class III/IV hemorrhagic shock.
Identifying shock prioritizes the 'Circulation' step in the primary trauma survey.
2
Identify the source of the hemorrhage.
Pelvic instability is present, and the FAST exam is negative.
A negative FAST makes intra-abdominal hemorrhage unlikely, localizing the life-threatening bleed to the retroperitoneal space due to the pelvic ring disruption.
3
Determine the appropriate initial intervention.
Application of a circumferential pelvic binder.
A pelvic binder quickly reduces the internal volume of the pelvis, which decreases venous bleeding from the presacral plexus and helps stabilize forming hemostatic clots.

Key Concept

Mechanical stabilization with a pelvic binder is the initial management step for hemodynamically unstable patients with unstable pelvic ring fractures.
Question 6Question →

Match each clinical presentation to the most likely pediatric orthopedic diagnosis.

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Items

A 13-year-old boy with a body mass index of 32 presents with an atraumatic limp and left knee pain. Examination shows the left hip is obligately externally rotated when actively flexed.
A 6-year-old boy presents with an insidious onset of a painless limp. Examination shows limited abduction and internal rotation of the hip. Radiographs reveal sclerosis and flattening of the proximal femoral epiphysis.
A 14-year-old basketball player presents with anterior knee pain worsened by jumping. Examination reveals localized tenderness and a bony prominence directly over the tibial tuberosity.
A 2-year-old girl refuses to use her right arm after being swung by her hands. The right upper extremity is held closely to her body in a pronated and extended position.
A 5-year-old boy presents with an acute onset limp and hip pain following a recent viral upper respiratory infection. He is afebrile, and his serum inflammatory markers are within normal limits.

Matches

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Answer

The correct pairings accurately match the clinical vignette of common pediatric musculoskeletal conditions to their respective diagnoses based on age, demographics, and key physical exam findings.
Each vignette represents the archetypal presentation of a pediatric orthopedic pathology. Recognition of defining features—such as obesity and referred knee pain in SCFE, or avascular necrosis changes in Legg-Calvé-Perthes—is required to form the correct matches.

Step-by-Step Solution

1
Analyze the presentation of the 13-year-old obese boy with knee pain and external hip rotation.
Matches with Slipped capital femoral epiphysis (SCFE).
SCFE predominantly affects overweight adolescents and frequently presents with referred knee pain and a pathognomonic loss of internal hip rotation.
2
Analyze the presentation of the 6-year-old boy with a painless limp and radiographic femoral head flattening.
Matches with Legg-Calvé-Perthes disease.
The age of onset (4 to 10 years), insidious progression, and avascular necrosis changes on x-ray are classic hallmarks of Legg-Calvé-Perthes disease.
3
Analyze the presentation of the 14-year-old athlete with tibial tubercle tenderness.
Matches with Osgood-Schlatter disease.
Mechanical overuse at the patellar tendon insertion site in growing athletes leads to Osgood-Schlatter disease.
4
Analyze the presentation of the 2-year-old girl with an immobile, pronated arm after being swung.
Matches with Radial head subluxation.
The mechanism of injury (axial traction) and the characteristic posture of the arm clearly indicate a nursemaid's elbow.
5
Analyze the presentation of the 5-year-old boy with acute hip pain following a viral illness and normal lab results.
Matches with Transient synovitis of the hip.
A benign, self-limiting inflammation of the hip joint following a recent viral infection without signs of septic arthritis points to transient synovitis.

Key Concept

Accurate diagnosis of pediatric orthopedic conditions relies heavily on the patient's age, mechanism of injury, associated demographics (e.g., obesity), and characteristic physical examination findings.
Estimated Time:1m 30s
Question 7Question →

A 26-year-old man is hospitalized after sustaining a closed mid-shaft fracture of the right femur in a motor vehicle collision. Due to multiple life-threatening cases in the operating room, his fracture is temporarily managed with skeletal traction. On the second day of hospitalization, he becomes acutely confused and agitated. His temperature is 38.3°C (100.9°F), blood pressure is 118/76 mmHg, heart rate is 122/min, and respiratory rate is 28/min. Pulse oximetry shows an oxygen saturation of 86% on room air. Physical examination reveals a non-blanching petechial rash over the anterior chest, axillae, and conjunctivae. Lung auscultation demonstrates bilateral scattered crackles. Which of the following interventions during the initial 24 hours of admission would have most effectively decreased the risk of this patient's current complication?

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Answer: Early operative internal fixation of the femur fracture

Answer

Early operative internal fixation of the femur fracture
The correct intervention is early operative internal fixation. This patient is experiencing Fat Embolism Syndrome (FES), characterized by the classic triad of respiratory distress, neurological changes, and a petechial rash occurring 24 to 72 hours after a major long bone (femur) fracture. The pathophysiology involves marrow fat droplets entering the venous circulation due to increased intramedullary pressure and tearing of venules at the fracture site. Early operative stabilization (e.g., open reduction and internal fixation) is the most proven method to reduce the incidence of FES because it eliminates ongoing fracture mobility and minimizes further marrow fat release.

Step-by-Step Solution

1
Analyze the clinical presentation to establish a diagnosis.
The patient exhibits the classic triad of hypoxemia, neurological abnormalities (confusion and agitation), and a petechial rash 24 to 48 hours after a long bone fracture, confirming Fat Embolism Syndrome (FES).
Accurate identification of the complication is necessary to determine what prophylactic measure was missed.
2
Identify the underlying pathophysiology of the diagnosed condition.
FES is caused by the release of fat droplets from the bone marrow into the systemic venous circulation, leading to microvascular occlusion and inflammation.
Understanding the mechanism highlights how the condition could have been prevented.
3
Determine the most effective preventative intervention.
Early operative stabilization of the fracture minimizes bone movement and marrow pressure, preventing further fat embolization.
This establishes early internal fixation as the correct preventative measure.

Key Concept

Fat Embolism Syndrome (FES) presents with a triad of respiratory distress, neurological dysfunction, and a petechial rash following long bone fractures. Early operative stabilization of the fracture is the most effective method for prevention.
Estimated Time:1m 15s
Question 8Question →

A 32-year-old man is evaluated by the surgical team for severe right lower extremity pain. Twelve hours ago, he underwent an uncomplicated open reduction and internal fixation of a comminuted right tibial shaft fracture sustained in a mountain biking accident. The patient reports that over the past two hours, the pain has become agonizing and is no longer relieved by his scheduled doses of intravenous morphine. His temperature is 37.2∘C37.2^\circ\text{C} (99.0∘F99.0^\circ\text{F}), blood pressure is 138/86 mmHg138/86\text{ mmHg}, and heart rate is 112/min112\text{/min}. On physical examination, the right anterolateral leg appears tense and significantly swollen compared to the contralateral limb. The overlying skin is shiny. Passive plantar flexion of the right ankle and toes elicits a dramatic increase in pain. Capillary refill time is 2 seconds, and the dorsalis pedis and posterior tibial pulses are palpable bilaterally. Which of the following is the most appropriate next step in management?

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Answer: Preparation for immediate fasciotomy

Answer

Preparation for immediate fasciotomy
The correct action is to prepare the patient for immediate surgical decompression. The patient's presentation of escalating pain that is out of proportion to the injury, a tense limb, and excruciating pain upon passive stretching of the affected muscles is highly specific for acute compartment syndrome (ACS). ACS occurs when increased pressure within an enclosed fascial space compromises tissue perfusion. The anterior compartment of the lower leg is most commonly affected, particularly following tibial fractures. Crucially, arterial pulses (dorsalis pedis) and capillary refill often remain normal until the latest stages of the syndrome, because the tissue pressure required to cause ischemia is lower than systolic arterial pressure. When the diagnosis is clinically evident, immediate fasciotomy is indicated to prevent irreversible muscle and nerve necrosis.

Step-by-Step Solution

1
Analyze the timeline and presenting symptoms
The patient is 12 hours postoperative from a tibial fracture repair and presents with pain out of proportion to the injury, accompanied by a tense, swollen leg.
Tibial shaft fractures and their surgical repairs are the most common causes of acute compartment syndrome (ACS).
2
Interpret the physical examination findings
Passive plantar flexion stretches the anterior compartment musculature, eliciting severe pain. Pulses and capillary refill remain intact.
Pain on passive stretch is the most sensitive early sign of ACS. Preserved pulses do not rule out the diagnosis, as arterial occlusion is a late and ominous finding.
3
Determine the necessary intervention based on clinical diagnosis
The clinical picture is diagnostic for acute compartment syndrome, requiring emergent surgical decompression.
Delaying treatment for imaging or applying conservative measures in clinically obvious ACS leads to irreversible tissue necrosis within hours.

Key Concept

Acute compartment syndrome is a surgical emergency characterized by pain out of proportion to injury and extreme pain on passive muscle stretch; it requires immediate fasciotomy, and palpable distal pulses do not rule out the diagnosis.
Estimated Time:1m 15s
Question 9Question →

A pediatric orthopedic clinic evaluates several patients with lower extremity complaints. Match each patient's clinical presentation with the most likely underlying diagnosis.

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Items

A 13-year-old boy with a BMI of 33 kg/m² presents with several weeks of left knee pain and a limp. Passive flexion of the left hip results in obligatory external rotation.
A 6-year-old boy is evaluated for a limp that has worsened over the past three months. He has limited abduction and internal rotation of the right hip. Radiographs show fragmentation of the femoral head.
A 4-year-old boy develops acute-onset right hip pain and refuses to bear weight 1 week after a viral upper respiratory infection. Vital signs and inflammatory markers are within normal limits.
A 14-year-old male basketball player presents with anterior knee pain exacerbated by jumping. Examination shows a prominent and tender tibial tubercle.

Matches

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Answer

The 13-year-old obese boy matches with Slipped capital femoral epiphysis; the 6-year-old with femoral head fragmentation matches with Legg-Calvé-Perthes disease; the 4-year-old post-viral patient matches with Transient synovitis of the hip; and the 14-year-old basketball player matches with Osgood-Schlatter disease.
Each clinical presentation contains classic demographic and physical exam hallmarks that uniquely identify the underlying pediatric orthopedic condition without overlapping features. SCFE presents with obligatory external rotation; Legg-Calvé-Perthes with avascular necrosis in younger boys; transient synovitis with acute post-viral pain and normal labs; and Osgood-Schlatter with tibial tubercle tenderness in jumping athletes.

Step-by-Step Solution

1
Analyze the demographic and physical exam findings for the 13-year-old boy.
The patient is an obese adolescent presenting with obligatory external rotation of the hip during passive flexion.
This physical exam finding, coupled with the patient's age and body habitus, is pathognomonic for slipped capital femoral epiphysis (SCFE).
2
Evaluate the clinical course and radiologic findings of the 6-year-old boy.
The chronic nature of the limp and fragmentation of the femoral head on radiographs indicate avascular necrosis.
Idiopathic avascular necrosis of the femoral head in this age group defines Legg-Calvé-Perthes disease.
3
Examine the history of the 4-year-old boy.
Acute hip pain following a viral illness with completely normal inflammatory markers.
The normal lab results rule out septic arthritis, making the benign, self-limiting condition of transient synovitis the correct diagnosis.
4
Assess the mechanism of injury and physical exam in the 14-year-old athlete.
Anterior knee pain during jumping activities with pinpoint tenderness over the tibial tubercle.
This is the classic presentation of a traction apophysitis of the tibial tubercle, known as Osgood-Schlatter disease.

Key Concept

Differentiating pediatric lower extremity pathologies based on patient age, body habitus, acuity of onset, and classic physical examination findings.
Estimated Time:1m 30s
Question 10Question →

A 6-year-old girl is evaluated in the emergency department after falling from a trampoline onto her outstretched left arm. She is in significant distress and refuses to move her upper extremity. Vital signs are stable. Physical examination reveals a gross S-shaped deformity and severe swelling of the left elbow. The skin over the antecubital fossa is intact without puckering. Capillary refill in the left fingers is 1.5 seconds, and the hand is warm and pink. However, the left radial pulse is completely absent to palpation and cannot be detected with a hand-held Doppler device. Neurologic examination reveals an inability to actively flex the interphalangeal joint of the left thumb, but she is able to extend her wrist and fingers against resistance. Radiographs demonstrate a completely displaced, extension-type supracondylar fracture of the distal humerus. The patient is emergently taken to the operating room, where an anatomic closed reduction and percutaneous pinning are performed. Intraoperative fluoroscopy confirms perfect alignment and stable fixation. Immediate post-procedural evaluation reveals that the left hand remains warm and pink with a capillary refill of 1.5 seconds. However, the radial pulse remains undetectable by Doppler. Which of the following is the most appropriate next step in the management of this patient?

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Answer: Admit the patient for continuous clinical observation and neurovascular monitoring

Answer

The correct answer is to admit the patient for continuous clinical observation and neurovascular monitoring.
The correct answer is to admit the patient for continuous clinical observation. This patient presents with a 'pink, pulseless' hand after anatomic closed reduction and percutaneous pinning of a displaced supracondylar humerus fracture. The brachial artery is highly susceptible to spasm, kinking, or entrapment in these fractures. Following reduction, if the radial pulse remains absent but the hand is well-perfused (warm, pink, capillary refill under 2 seconds), it indicates that the rich collateral circulation around the elbow is providing sufficient blood flow to maintain limb viability. Current orthopedic guidelines recommend close clinical observation for a pink, pulseless hand post-reduction, as pulses often return within 24 to 48 hours as vasospasm resolves. Immediate vascular exploration is only indicated for an ischemic ('pale, pulseless') hand that does not improve after fracture reduction.

Step-by-Step Solution

1
Evaluate the post-reduction vascular status.
The left hand is warm and pink with brisk capillary refill, but the radial pulse remains absent (a 'pink, pulseless' hand).
To determine the adequacy of distal limb perfusion following the surgical intervention.
2
Determine the indication for vascular intervention.
Immediate vascular exploration is not indicated.
The presence of a warm, pink hand with capillary refill under 2 seconds indicates that collateral circulation around the elbow is providing sufficient blood flow for limb viability, despite the absent radial pulse.
3
Formulate the next best step in management.
Select close clinical observation.
In a well-perfused but pulseless extremity following anatomic reduction of a supracondylar fracture, conservative management with continuous monitoring is the standard of care as pulses typically return gradually as arterial spasm resolves.

Key Concept

Management of a pink, pulseless hand following reduction of a pediatric supracondylar humerus fracture.
Question 11Question →

A 24-year-old male presents to the emergency department supporting his right arm after an awkward fall during a recreational rugby match. He is in significant pain, and his right shoulder has a visibly 'squared-off' appearance. He complains of numbness over the lateral aspect of his shoulder. Arrange the following steps in the most appropriate chronological sequence for the acute management of this patient's presentation.

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Answer

The correct sequence is: Initial neurovascular examination, followed by pre-reduction radiographs, then closed reduction, and finally post-reduction reassessment and imaging.
The fundamental principle of managing suspected joint dislocations is to first assess and document the baseline neurovascular status. Next, radiographs must be obtained to confirm the diagnosis and rule out fractures. Once the joint is cleared for manipulation, closed reduction is performed. Finally, post-reduction neurovascular assessment and imaging are mandatory to confirm procedural success and rule out iatrogenic complications.

Step-by-Step Solution

1
Determine the very first crucial step in evaluating a trauma patient with an obvious joint deformity.
Perform a focused neurovascular examination.
Establishing a baseline neurovascular status is critical before any intervention. It documents pre-existing nerve or vascular injuries (such as an axillary nerve injury, common in anterior shoulder dislocations) and prevents confusion later if deficits are found post-reduction.
2
Identify the next step required before attempting to correct the anatomical deformity.
Obtain pre-reduction radiographs.
Imaging is absolutely necessary to confirm the exact type of dislocation and to rule out concomitant fractures (e.g., surgical neck fracture of the humerus) that could be displaced during manipulation and turn a closed reduction into a surgical emergency.
3
Determine the primary therapeutic intervention once the diagnosis is confirmed and fractures are ruled out.
Administer procedural sedation and perform closed reduction.
Prompt reduction is performed under adequate analgesia or sedation to relieve the patient's pain, restore joint anatomy, and prevent long-term articular cartilage or neurovascular damage.
4
Identify the required post-procedure safety and verification protocol.
Reassess neurovascular status, apply a sling, and obtain post-reduction radiographs.
Reassessment ensures that no iatrogenic nerve or vessel injury occurred during the manipulation. Immobilization protects the unstable joint, and repeat imaging verifies a successful, concentric reduction.

Key Concept

The standard sequence of acute management for suspected joint dislocations follows the principles of clinical assessment, diagnostic imaging, therapeutic reduction, and post-procedural reassessment.
Question 12Question →

Match each clinical and radiographic vignette with the most likely primary bone tumor diagnosis.

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Items

A 15-year-old boy presents with progressive distal thigh pain. Labs show elevated alkaline phosphatase. Radiographs reveal a destructive, mixed lytic and blastic metaphyseal lesion with elevation of the periosteum at the margins.
A 13-year-old boy presents with localized mid-thigh pain, low-grade fever, and leukocytosis. Radiographs show a permeative diaphyseal lesion with concentric, lamellated layers of reactive bone formation.
A 30-year-old woman presents with worsening knee pain with weight-bearing. Radiographs show a purely osteolytic, eccentric lesion extending into the proximal tibial epiphysis without a sclerotic rim.
A 22-year-old man presents with dull thigh pain that wakes him from sleep but is completely relieved by ibuprofen. Radiographs demonstrate a small, circumscribed radiolucent nidus surrounded by dense reactive cortical sclerosis.

Matches

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Answer

The correct pairings match each specific bone tumor presentation with its classic demographic, anatomical location, and radiographic features.
Each tumor has a distinct predilection for patient age and long bone anatomy. Osteosarcoma arises in the metaphysis of teens; Ewing sarcoma in the diaphysis of children/teens; Giant cell tumors in the epiphysis of adults; and Osteoid osteomas present with NSAID-responsive night pain and a characteristic nidus.

Step-by-Step Solution

1
Analyze the first vignette for demographic and radiographic clues.
Identified adolescent age, metaphyseal location, mixed lytic/blastic pattern, and elevated periosteum (Codman triangle), which points to Osteosarcoma.
Osteosarcoma is the most common primary malignant bone tumor in adolescents and classically occurs in the metaphysis of long bones.
2
Analyze the second vignette focusing on systemic symptoms and location.
Identified diaphyseal location, 'onion-skin' periosteal reaction, and constitutional symptoms mimicking osteomyelitis, indicating Ewing sarcoma.
Ewing sarcoma often presents with fever and leukocytosis and is classically found in the diaphysis of long bones.
3
Evaluate the third vignette for age and specific bone region.
Identified an adult patient (post-physeal closure) with an epiphyseal, eccentric, lytic lesion, which corresponds to a Giant cell tumor of bone.
Giant cell tumors typically arise in the epiphysis of long bones in young adults between 20 and 40 years of age.
4
Review the fourth vignette for classic symptom descriptors.
Identified nocturnal pain completely relieved by NSAIDs and a radiolucent nidus with surrounding sclerosis, confirming Osteoid osteoma.
Prostaglandin-producing osteoid osteomas cause night pain that responds dramatically to prostaglandin inhibitors like ibuprofen.

Key Concept

Primary Bone Tumors: Demographics, Anatomical Location, and Radiographic Features
Estimated Time:2m 0s
Question 13Question →

A 32-year-old man is brought to the emergency department after a high-speed motorcycle collision. His airway is patent, breathing is unlabored, and he is hemodynamically stable. Examination reveals a grossly deformed right lower leg with a 4-cm anteromedial laceration through which fractured bone fragments are visible. The wound is oozing dark blood, but there is no pulsatile bleeding.

Arrange the following steps in the most appropriate sequential order for the acute management of this patient's orthopedic injury.

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Answer

The correct sequence of acute management is: 1) Assess neurovascular status, 2) Administer intravenous antibiotics and tetanus prophylaxis, 3) Apply a sterile dressing and splint, 4) Obtain radiographs, and 5) Transfer to the operating room for definitive surgical care.
In the acute management of an open fracture, following the primary survey (ABCDEs), the immediate next step is to evaluate the limb's neurovascular status to establish a baseline. Because the risk of infection is the most significant complication, intravenous antibiotics must be given as quickly as possible (typically a first-generation cephalosporin, with or without an aminoglycoside depending on wound severity). After these immediate steps, the wound should be covered with a sterile dressing and splinted to prevent further soft tissue injury. Only when the limb is stabilized should the patient be sent for radiographs. Finally, the patient is taken to the operating room for definitive surgical irrigation, debridement, and stabilization.

Step-by-Step Solution

1
Perform a primary and secondary survey, immediately assessing the distal neurovascular status of the injured limb.
Baseline vascular (pulses, capillary refill) and neurologic (motor, sensory) function is documented.
Manipulation or splinting can alter the neurovascular exam; a baseline is essential to identify immediate surgical emergencies like arterial transection.
2
Order and administer broad-spectrum intravenous antibiotics and update tetanus prophylaxis.
Systemic antimicrobial coverage is achieved early in the patient's presentation.
Time-to-antibiotic administration is the most critical prognostic factor in preventing deep bone infection (osteomyelitis) in open fractures.
3
Cover the open wound with a sterile saline dressing and apply a temporary splint.
The wound is protected from hospital flora and the unstable bone ends are immobilized.
Immobilization controls pain, minimizes ongoing soft tissue damage, and protects the limb during subsequent movements for imaging.
4
Transport the patient for orthogonal plain radiographs of the tibia, including the knee and ankle.
The exact fracture morphology and joint involvement are visualized.
Imaging is required for surgical planning, but must only be done after the limb is safely splinted to avoid displacing bone fragments.
5
Transfer the patient to the operating room.
Formal surgical irrigation, debridement, and fracture fixation are performed.
This is the definitive treatment for open fractures to ensure a clean wound bed and provide skeletal stability.

Key Concept

The initial management of an open fracture prioritizes early neurovascular assessment and prompt antibiotic administration before splinting, imaging, and definitive surgical care.
Estimated Time:1m 30s
Question 14Question →

A 9-year-old boy is brought to the emergency department by his parents due to a 3-day history of worsening left leg pain and fever. The parents report that he initially started limping but now refuses to bear any weight on the left leg. There is no history of recent trauma, tick bites, or travel. His temperature is 38.8°C (101.8°F), blood pressure is 105/65 mmHg, heart rate is 115/min, and respiratory rate is 18/min. On physical examination, the child is ill-appearing. There is exquisite point tenderness, localized warmth, and mild swelling over the proximal anterior aspect of the left tibia. Range of motion of the left knee and ankle is fully preserved and does not elicit pain. Laboratory studies reveal a leukocyte count of 15,500/mm³ (85% neutrophils), an erythrocyte sedimentation rate (ESR) of 65 mm/hr, and a C-reactive protein (CRP) level of 82 mg/L. Plain radiographs of the left tibia and knee show no obvious abnormalities. Which of the following is the most appropriate next step in management?

Show answer & explanation

Answer: Obtain magnetic resonance imaging of the left lower extremity

Answer

Obtain magnetic resonance imaging of the left lower extremity
The most appropriate next step in evaluation is to obtain a magnetic resonance imaging (MRI) scan of the lower extremity. This pediatric patient presents with localized bone pain, high fever, and elevated inflammatory markers (WBC, ESR, CRP), which is highly suspicious for acute hematogenous osteomyelitis. In children, this infection typically seeds in the highly vascular metaphysis of long bones. Plain radiographs are typically normal during the first 10 to 14 days of infection because approximately 30% to 50% of bone mineral must be lost before lytic changes become radiographically apparent. MRI, with and without contrast, is the most sensitive and specific imaging modality for early osteomyelitis, capable of detecting bone marrow edema and surrounding soft tissue involvement (e.g., subperiosteal abscess) within 3 to 5 days of onset.

Step-by-Step Solution

1
Analyze the clinical presentation.
The patient is a 9-year-old boy with acute onset of localized proximal tibial pain, refusal to bear weight, high fever, and no history of trauma.
Identifying these classic symptoms strongly points to acute hematogenous osteomyelitis.
2
Evaluate the laboratory and imaging findings.
Elevated WBC, ESR, and CRP confirm an acute inflammatory/infectious process. Normal plain radiographs are noted.
Plain radiographs are typically normal in the first 10-14 days of osteomyelitis because significant bone demineralization has not yet occurred, making further imaging necessary.
3
Determine the next best step in management.
MRI of the affected limb is indicated.
In a hemodynamically stable patient, MRI is the gold standard to confirm early bone marrow edema and evaluate the extent of the infection before initiating antibiotic therapy.

Key Concept

Early acute hematogenous osteomyelitis typically presents with normal plain radiographs; MRI is the most sensitive imaging modality and is the next best step for definitive diagnosis in a stable patient.
Question 15Question →

Match each clinical vignette describing an orthopedic injury with the most appropriate definitive management step.

Click a left item, then click its matching right item

Items

A 32-year-old man with a closed tibial fracture develops excruciating leg pain and paresthesias; his intracompartmental pressure is 45 mmHg and his diastolic blood pressure is 70 mmHg.
A 72-year-old woman with a history of severe, symptomatic left hip osteoarthritis sustains a displaced, completely intracapsular left femoral neck fracture following a mechanical fall.
A 21-year-old male sustains a completely displaced mid-shaft clavicle fracture with 3 cm of shortening and severe skin tenting causing marked blanching of the overlying soft tissue.
A 28-year-old man presents with persistent anatomic snuffbox tenderness 8 months after a wrist injury; MRI demonstrates a scaphoid waist nonunion with avascular necrosis of the proximal pole.

Matches

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Answer

The tibial fracture patient with elevated compartment pressure matches with immediate four-compartment fasciotomy. The elderly patient with a femoral neck fracture and baseline severe osteoarthritis matches with total hip arthroplasty. The clavicle fracture with skin blanching matches with urgent open reduction and internal fixation. The scaphoid nonunion with avascular necrosis matches with vascularized bone grafting and internal fixation.
Each pairing relies on identifying absolute indications that deviate from standard or less invasive management. The tibial fracture patient has acute compartment syndrome (delta pressure < 30 mmHg) requiring fasciotomy. The older patient with a femoral neck fracture and baseline osteoarthritis requires a total hip arthroplasty rather than a hemiarthroplasty to address the arthritic acetabulum. The clavicle fracture patient has impending skin necrosis, an absolute indication for ORIF. Finally, scaphoid nonunion with proximal pole AVN requires a vascularized bone graft due to the compromised retrograde blood supply.

Step-by-Step Solution

1
Calculate the delta pressure for the patient with the closed tibial fracture.
The delta pressure is 25 mmHg (70−45=2570 - 45 = 25).
A delta pressure of less than 30 mmHg is the diagnostic threshold for acute compartment syndrome, which dictates the need for emergent surgical decompression.
2
Analyze the optimal surgical approach for the 72-year-old with a displaced femoral neck fracture.
Identify that total hip arthroplasty (THA) is required over hemiarthroplasty.
While hemiarthroplasty is standard for many elderly patients with this specific fracture, the presence of severe pre-existing osteoarthritis dictates THA to address both the fracture and the diseased native acetabulum.
3
Identify the complication risk in the patient with the displaced clavicle fracture and skin blanching.
Recognize an impending open fracture.
Severe skin tenting with blanching indicates ischemia of the overlying skin; if left untreated, the skin will necrose, converting a closed fracture into an open one, making urgent ORIF necessary.
4
Determine the graft requirement for a scaphoid nonunion with proximal pole avascular necrosis (AVN).
Select vascularized bone grafting and internal fixation.
The proximal pole of the scaphoid has a tenuous retrograde blood supply. Once AVN occurs, a standard non-vascularized bone graft will fail to integrate, requiring a vascularized graft to restore local perfusion and promote osseous union.

Key Concept

Absolute surgical indications and advanced orthopedic management strategies for specific trauma complications and fracture patterns.
Estimated Time:2m 0s
Question 16Question →

Match each clinical presentation of lower extremity pain with its most likely orthopedic diagnosis.

Click a left item, then click its matching right item

Items

Anterior knee pain that worsens with descending stairs and is reproducible by patellar compression during quadriceps contraction.
Stabbing heel pain that is most severe with the first few steps in the morning and gradually improves with ambulation.
Lateral knee pain in a distance runner, associated with focal tenderness roughly 2-3 cm proximal to the lateral joint line.
Burning forefoot pain and numbness accompanied by a painful click when the metatarsal heads are squeezed together.

Matches

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Answer

The presentations map to Patellofemoral pain syndrome (anterior knee with compression pain), Plantar fasciitis (morning heel pain), Iliotibial band syndrome (lateral knee pain in runners), and Morton neuroma (forefoot pain with clicking).
Each condition has a classic hallmark presentation. Patellofemoral pain syndrome affects the anterior knee under the patella. Plantar fasciitis produces hallmark morning heel pain. Iliotibial band syndrome affects the lateral knee near the epicondyle in runners. Morton neuroma produces neuropathic forefoot pain with a palpable click (Mulder sign).

Step-by-Step Solution

1
Analyze the location and character of the pain described in each clinical vignette.
Identified four distinct anatomical localizations: anterior knee, plantar heel, lateral knee, and plantar forefoot.
Accurate anatomical localization is the critical first step in evaluating and diagnosing musculoskeletal pathology.
2
Correlate the provocative factors and physical exam maneuvers with specific structural pathologies.
Patellar compression points to the patellofemoral joint; morning first-step pain points to the plantar fascia; lateral epicondyle tenderness points to the IT band; and a painful click (Mulder sign) points to an interdigital neuroma.
Provocative tests confirm the specific tissue involved, distinguishing between adjacent structures (e.g., distinguishing IT band friction from a lateral meniscus tear).

Key Concept

Clinical diagnosis of common outpatient lower extremity orthopedic conditions relies heavily on specific anatomical pain localization and classic provocative clinical findings.
Question 17Question →

Match the following acute upper extremity orthopedic injuries with their most characteristic physical examination findings and anatomical disruptions.

Click a left item, then click its matching right item

Items

Scaphoid fracture
Lunate dislocation
Monteggia fracture-dislocation
Galeazzi fracture-dislocation

Matches

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Answer

Scaphoid fracture matches with anatomic snuffbox tenderness. Lunate dislocation matches with volar swelling and median nerve paresthesias. Monteggia fracture-dislocation matches with proximal forearm deformity and radiocapitellar dislocation. Galeazzi fracture-dislocation matches with distal forearm deformity and prominent ulnar head.
Each named injury corresponds to a distinct anatomical disruption. The scaphoid sits at the base of the anatomic snuffbox, making this the primary site of tenderness when fractured. The lunate articulates directly above the carpal tunnel; its volar dislocation inherently compromises the median nerve space. Eponymous forearm fractures are defined by their specific bone and joint involvement: Monteggia affects the proximal ulna and radial head, while Galeazzi affects the distal radius and distal radioulnar joint.

Step-by-Step Solution

1
Analyze the carpal bone injuries and their specific localizations.
Identify that the scaphoid correlates with the anatomic snuffbox, while the lunate is centrally located and can compress the median nerve if dislocated volarly.
Differentiating carpal injuries relies on identifying the exact site of tenderness or secondary nerve compression.
2
Distinguish between the eponymous forearm fracture-dislocations.
Apply the MUGR mnemonic (Monteggia = Ulna fracture with proximal radial dislocation; Galeazzi = Radius fracture with distal ulnar dislocation).
These combined injuries are frequently confused; remembering the specific bone fractured helps identify which adjacent joint is dislocated.

Key Concept

Upper extremity orthopedic injuries have highly specific anatomical presentations, such as the MUGR pattern for forearm fracture-dislocations and distinct focal tenderness or nerve compression for carpal injuries.
Estimated Time:1m 0s
Question 18Question →

Match each of the following specific orthopedic injuries with its most commonly associated acute neurovascular deficit as assessed on physical examination.

Click a left item, then click its matching right item

Items

Midshaft humeral fracture
Anterior shoulder dislocation
Posterior hip dislocation
Supracondylar humerus fracture (anterolateral displacement)

Matches

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Answer

Midshaft humeral fracture matches with inability to extend the wrist and fingers. Anterior shoulder dislocation matches with loss of sensation over the lateral proximal arm. Posterior hip dislocation matches with weakness in ankle dorsiflexion. Supracondylar humerus fracture matches with inability to flex the distal joints of the thumb and index finger.
Each specific fracture or dislocation puts a predictable peripheral nerve at risk due to close anatomical proximity. The radial nerve is intimately associated with the midshaft humerus; the axillary nerve courses just inferior to the glenohumeral joint capsule; the sciatic nerve lies directly posterior to the hip joint; and the median/anterior interosseous nerve crosses anterior to the supracondylar region of the humerus.

Step-by-Step Solution

1
Identify the precise anatomical location and mechanism of each listed orthopedic injury.
Determined that the injuries involve the mid-diaphysis of the humerus, the anterior glenohumeral joint, the posterior acetabulum/femoral head, and the distal humerus metaphysis, respectively.
Understanding the exact location of the bony or ligamentous disruption is necessary to identify adjacent structures at risk.
2
Correlate the injury location with adjacent peripheral nerves and their expected downstream motor and sensory deficits.
Mapped the midshaft humerus to the radial nerve (wrist extension), the anterior shoulder to the axillary nerve (lateral arm sensation), the posterior hip to the sciatic nerve (ankle dorsiflexion via the common peroneal nerve branch), and the anterolaterally displaced distal humerus to the anterior interosseous nerve (distal thumb/index flexion).
This establishes the clinical presentation expected for each specific neurovascular complication on physical examination.

Key Concept

Neurovascular complications of common orthopedic fractures and dislocations
Estimated Time:1m 30s
Question 19Question →

A 34-year-old man is brought to the emergency department for severe right shoulder pain. Thirty minutes ago, he experienced a witnessed generalized tonic-clonic seizure that lasted 2 minutes. On physical examination, the right arm is held firmly in adduction and internal rotation. The patient actively resists any attempt at passive external rotation due to severe pain. The radial pulse is 2+ and sensation is intact to light touch over the deltoid and distal fingers. An initial anteroposterior (AP) radiograph of the right shoulder reveals a symmetric, rounded appearance of the humeral head with no obvious fracture lines. Which of the following is the most appropriate next step in the management of this patient?

Show answer & explanation

Answer: Obtain an axillary or scapular Y-view radiograph of the right shoulder

Answer

Obtain an axillary or scapular Y-view radiograph of the right shoulder
The correct next step is to obtain an axillary or scapular Y-view radiograph. This patient's clinical presentation—arm locked in internal rotation and adduction following a seizure—is highly characteristic of a posterior shoulder dislocation. The intense muscle contractions during a generalized seizure cause the strong internal rotators (latissimus dorsi, pectoralis major, subscapularis) to overpower the external rotators (infraspinatus, teres minor). Posterior dislocations are frequently missed on standard anteroposterior (AP) radiographs because the humeral head rotates internally, masking the typical overlap seen in anterior dislocations and creating a symmetric, rounded appearance known as the 'lightbulb sign'. Therefore, a dedicated orthogonal view (such as an axillary or scapular Y-view) is strictly required to verify the posterior displacement and identify any associated defects (e.g., reverse Hill-Sachs lesion) prior to attempting closed reduction.

Step-by-Step Solution

1
Analyze the mechanism of injury and physical exam findings.
The patient had a seizure, followed by the arm being locked in adduction and internal rotation with severe pain on external rotation.
Violent muscle contractions during a seizure or electrocution overpower external rotators, classically causing a posterior shoulder dislocation.
2
Evaluate the initial imaging provided.
The AP radiograph shows a symmetric, rounded humeral head (the 'lightbulb sign') and no obvious fracture.
Posterior dislocations are notorious for looking deceptively normal on standard AP views because the humeral head rotates internally, causing the articular surface to face posteriorly and appear symmetric on a 2D anterior-to-posterior plane.
3
Determine the necessary diagnostic intervention to confirm the suspected diagnosis.
An orthogonal plain radiographic view (axillary or scapular Y-view) must be obtained.
An orthogonal view will definitively show the humeral head displaced posteriorly relative to the glenoid fossa and help rule out associated fractures (such as a reverse Hill-Sachs lesion) before any reduction is attempted.

Key Concept

Posterior Shoulder Dislocation Diagnosis
Question 20Question →

A 25-year-old man is brought to the emergency department after a motorcycle collision. Examination reveals an obvious deformity of the right lower leg with a 4-centimeter laceration over the anterior tibia, exposing the underlying fractured bone. Distal pulses are palpable, and the patient has intact sensation in the foot. Arrange the following steps in the most appropriate chronological sequence for the acute management of this patient's open fracture.

Drag items to arrange them in the correct order

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Answer

The correct sequence for managing an open fracture is to first perform a primary trauma survey (ABCs), then administer intravenous antibiotics and tetanus prophylaxis, followed by bedside irrigation and temporary splinting, and finally transfer to the operating room for definitive debridement and fixation.
This sequence strictly adheres to standard trauma and orthopedic protocols. Patient survival supersedes limb salvage, mandating the ABCs first. Once the patient is deemed stable, preventing deep bone infection becomes the priority, making early IV antibiotic administration the next critical step. Following medical prophylaxis, the limb is provisionally realigned and splinted to protect soft tissues and relieve pain. Finally, the patient is transferred to the operating room for definitive surgical irrigation, debridement, and skeletal fixation.

Step-by-Step Solution

1
Ensure patient survival and hemodynamic stability.
Primary trauma survey (ABCs) is completed.
Life-threatening injuries must be identified and managed before addressing extremity injuries.
2
Provide early medical prophylaxis against infection.
Intravenous antibiotics and tetanus prophylaxis are administered.
Time to antibiotic administration is the single most important factor in preventing osteomyelitis in open fractures.
3
Stabilize the extremity temporarily.
Bedside irrigation, reduction, and temporary splinting are performed.
This removes gross debris, restores neurovascular alignment, and prevents further soft tissue damage during transport.
4
Provide definitive surgical care.
Patient is taken to the OR for formal debridement and fixation.
Definitive fracture stabilization and thorough excision of non-viable tissue require a sterile operating room environment.

Key Concept

The acute management of open fractures prioritizes patient survival (ABCs), followed immediately by infection prevention (early IV antibiotics), then provisional limb stabilization, and finally definitive operative debridement and fixation.
Estimated Time:1m 0s
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Orthopedic Surgery Practice Questions — USMLE Step 2 CK | Examkin